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Jeremy M. Simon

RWTH Aachen University · US
🔎 Find collaborators in Molecular Biology · Genetics →
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Area of research
Molecular Biology · Genetics
Research interest
Research interests include Epigenetics and DNA Methylation, Genetics and Neurodevelopmental Disorders, Chromatin Remodeling and Cancer, and Cancer Mechanisms and Therapy.
h-index
39
citations
11,395
works
215
NIH funding
primary concept
email

Recent publications

A neoantigen vaccine generates antitumour immunity in renal cell carcinoma
Nature 2025cited by 155position: middledoi
BBOX1 restrains TBK1-mTORC1 oncogenic signaling in clear cell renal cell carcinoma.
2025cited by 7position: contributordoi
Spt6-Spn1 interaction is required for RNA polymerase II association and precise nucleosome positioning along transcribed genes
Journal of Biological Chemistry 2025cited by 3position: middledoi
Loss of UBE3A impacts both neuronal and non-neuronal cells in human cerebral organoids.
2025cited by 2position: contributordoi
SWI/SNF ATPase silenced HLF potentiates lung metastasis in solid cancers.
2025cited by 2position: contributordoi
Activating NRF2<sup>E79Q</sup> mutation alters the differentiation of human non-small cell lung cancer.
2025cited by 0position: contributordoi
Activating NRF2E79Q mutation alters the differentiation of human non-small cell lung cancer
2025cited by 0position: middledoi
2'3'-cGAMP interactome identifies 2'3'-cGAMP/Rab18/FosB signaling in cell migration control independent of innate immunity.
2024cited by 4position: contributordoi
Septo-dentate gyrus cholinergic circuits modulate function and morphogenesis of adult neural stem cells through granule cell intermediaries.
2024cited by 3position: contributordoi
Author Correction: An oncogenic Ezh2 mutation induces tumors through global redistribution of histone 3 lysine 27 trimethylation.
2024cited by 2position: contributordoi
DAB2IP loss in luminal a breast cancer leads to NF-κB-associated aggressive oncogenic phenotypes.
2024cited by 1position: middledoi
Distinct roles for canonical and variant histone H3 lysine-36 in Polycomb silencing.
2023cited by 30position: contributordoi
RNA-binding deficient TDP-43 drives cognitive decline in a mouse model of TDP-43 proteinopathy.
2023cited by 16position: contributordoi
RNA-binding deficient TDP-43 drives cognitive decline in a mouse model of TDP-43 proteinopathy
eLife 2023cited by 15position: middledoi
RNA-binding deficient TDP-43 drives cognitive decline in a mouse model of TDP-43 proteinopathy
2023cited by 2position: middledoi
RNA-binding deficient TDP-43 drives cognitive decline in a mouse model of TDP-43 proteinopathy
2023cited by 1position: middledoi
Supplementary Table S1 from Identification of BBOX1 as a Therapeutic Target in Triple-Negative Breast Cancer
2023cited by 0position: middledoi
Data from Identification of BBOX1 as a Therapeutic Target in Triple-Negative Breast Cancer
2023cited by 0position: middledoi
Supplementary Table S1 from Identification of BBOX1 as a Therapeutic Target in Triple-Negative Breast Cancer
2023cited by 0position: middledoi
Data from Identification of BBOX1 as a Therapeutic Target in Triple-Negative Breast Cancer
2023cited by 0position: middledoi
Supplementary Data from Identification of BBOX1 as a Therapeutic Target in Triple-Negative Breast Cancer
2023cited by 0position: middledoi
Supplementary Data from Identification of BBOX1 as a Therapeutic Target in Triple-Negative Breast Cancer
2023cited by 0position: middledoi
Author Response: RNA-binding deficient TDP-43 drives cognitive decline in a mouse model of TDP-43 proteinopathy
2023cited by 0position: middledoi
Data from PBRM1 Inactivation Promotes Upregulation of Human Endogenous Retroviruses in a HIF-Dependent Manner
2023cited by 0position: middledoi
Data from PBRM1 Inactivation Promotes Upregulation of Human Endogenous Retroviruses in a HIF-Dependent Manner
2023cited by 0position: middledoi
Supplementary Figure from PBRM1 Inactivation Promotes Upregulation of Human Endogenous Retroviruses in a HIF-Dependent Manner
2023cited by 0position: middledoi
Supplementary Figure from PBRM1 Inactivation Promotes Upregulation of Human Endogenous Retroviruses in a HIF-Dependent Manner
2023cited by 0position: middledoi
Supplementary Data from PBRM1 Inactivation Promotes Upregulation of Human Endogenous Retroviruses in a HIF-Dependent Manner
2023cited by 0position: middledoi
Supplementary Data from PBRM1 Inactivation Promotes Upregulation of Human Endogenous Retroviruses in a HIF-Dependent Manner
2023cited by 0position: middledoi
RNA-binding deficient TDP-43 drives cognitive decline in a mouse model of TDP-43 proteinopathy
2023cited by 0position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Qing Zhang · Southwestern Medical Center15 papers (2019–2025)Austin Hepperla · University of North Carolina at Chapel Hill11 papers (2022–2025)Julie Necarsulmer · Pomona College10 papers (2022–2023)Todd J. Cohen · Neurology, Inc10 papers (2022–2023)Kathryn M. Harper · University of Guelph10 papers (2022–2023)Lianxin Hu · Southwestern Medical Center9 papers (2020–2025)Mark J. Zylka · University of North Carolina at Chapel Hill9 papers (2019–2022)Laura E. Herring · Communities In Schools of Orange County8 papers (2020–2024)Charles M. Perou · Oryzon Genomics (Spain)8 papers (2019–2023) · 8 papers (2019–2023)Ariana Marquez Gonzalez · University of North Carolina at Chapel Hill8 papers (2022–2023)Jason W. Locasale · North Central State College8 papers (2019–2023)Yan Peng · Robert Bosch (Germany)7 papers (2020–2023)William Y. Kim · Chapel Hill Carrboro City Schools7 papers (2022–2023)Mi Zhou · State Key Laboratory of Oral Diseases7 papers (2022–2023)Ian J. Davis · University of North Carolina at Chapel Hill7 papers (2022–2023)Giada Zurlo · Kedrion (Italy)7 papers (2020–2023)Chengheng Liao · Taipei Medical University6 papers (2020–2025)Benjamin D. Philpot · University of North Carolina at Chapel Hill5 papers (2020–2022)Hyojin Kim · Life Edit Therapeutics3 papers (2021–2022)
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