Area of research
Cell Biology · Epidemiology
Research interest
Research interests include Autophagy in Disease and Therapy, Cellular transport and secretion, Ubiquitin and proteasome pathways, and Endoplasmic Reticulum Stress and Disease.
Intercellular nanotube-mediated mitochondrial transfer enhances T cell metabolic fitness and antitumor efficacy
Simultaneous detection of membrane contact dynamics and associated Ca2+ signals by reversible chemogenetic reporters
A tripartite organelle platform links growth factor receptor signaling to mitochondrial metabolism
An intrinsic mechanism of metabolic tuning promotes cardiac resilience to stress
A tripartite organelle platform links growth factor receptor signaling to mitochondrial metabolism
TMX4-driven LINC complex disassembly and asymmetric autophagy of the nuclear envelope upon acute ER stress
Pathological mitophagy disrupts mitochondrial homeostasis in Leber’s hereditary optic neuropathy
Mitochondria-rough-ER contacts in the liver regulate systemic lipid homeostasis
A three-organelle complex made by wrappER contacts with peroxisomes and mitochondria responds to liver lipid flux changes
Impaired mitophagy links mitochondrial disease to epithelial stress in methylmalonyl-CoA mutase deficiency
Salivary gland macrophages and tissue-resident CD8 <sup>+</sup> T cells cooperate for homeostatic organ surveillance
Molecularly Distinct Clathrin-Coated Pits Differentially Impact EGFR Fate and Signaling
Advanced Fluorescent Polymer Probes for the Site-Specific Labeling of Proteins in Live Cells Using the HaloTag Technology
A selective <scp>ER</scp> ‐phagy exerts procollagen quality control via a Calnexin‐ <scp>FAM</scp> 134B complex
BAP1 regulates IP3R3-mediated Ca2+ flux to mitochondria suppressing cell transformation
Reticulon 3–dependent ER-PM contact sites control EGFR nonclathrin endocytosis
Autophagy regulates UBC9 levels during viral-mediated tumorigenesis
Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery
Optic Atrophy 1 Is Epistatic to the Core MICOS Component MIC60 in Mitochondrial Cristae Shape Control
PML at Mitochondria-Associated Membranes Is Critical for the Repression of Autophagy and Cancer Development
The leukodystrophy protein FAM126A (hyccin) regulates PtdIns(4)P synthesis at the plasma membrane
Reduced release probability prevents vesicle depletion and transmission failure at dynamin mutant synapses