Area of research
Molecular Biology · Public Health, Environmental and Occupational Health
Research interest
Research interests include Endocrinology, Cancer research, Biology, Medicine, Heart failure, and Internal medicine.
Impaired immune response mediated by prostaglandin E2 promotes severe COVID-19 disease
Neuraminidase-1 promotes heart failure after ischemia/reperfusion injury by affecting cardiomyocytes and invading monocytes/macrophages
Venetoclax and dexamethasone synergize with inotuzumab ozogamicin–induced DNA damage signaling in B-lineage ALL
Lipid nanoparticle-mediated siRNA delivery for safe targeting of human CML in vivo
In peripartum cardiomyopathy plasminogen activator inhibitor-1 is a potential new biomarker with controversial roles
Electrophysiological abnormalities in induced pluripotent stem cell‐derived cardiomyocytes generated from Duchenne muscular dystrophy patients
A positive feedback loop between IL-1β, LPS and NEU1 may promote atherosclerosis by enhancing a pro-inflammatory state in monocytes and macrophages
Insulin supplementation attenuates cancer-induced cardiomyopathy and slows tumor disease progression
NKL homeobox gene activities in hematopoietic stem cells, T-cell development and T-cell leukemia
Suppression of RUNX1/ETO oncogenic activity by a small molecule inhibitor of tetramerization
Low STAT3 expression sensitizes to toxic effects of β-adrenergic receptor stimulation in peripartum cardiomyopathy
Opposing roles of Akt and STAT3 in the protection of the maternal heart from peripartum stress
BCR-ABL Affects STAT5A and STAT5B Differentially
MicroRNA-146a is a therapeutic target and biomarker for peripartum cardiomyopathy
Differential expression of miR-17∼92 identifies BCL2 as a therapeutic target in BCR-ABL-positive B-lineage acute lymphoblastic leukemia
MicroRNA-20a Constrains p300-Driven Myocardial Angiogenic Transcription by Direct Targeting of p300
Interleukin-1 Assembles a Proangiogenic Signaling Module Consisting of Caveolin-1, Tumor Necrosis Factor Receptor–Associated Factor 6, p38–Mitogen-Activated Protein Kinase (MAPK), and MAPK-Activated Protein Kinase 2 in Endothelial Cells
Reduction of STAT3 expression induces mitochondrial dysfunction and autophagy in cardiac HL-1 cells