Area of research
Immunology · Oncology
Research interest
Research interests include Immune Cell Function and Interaction, Immunotherapy and Immune Responses, T-cell and B-cell Immunology, and CAR-T cell therapy research.
Semaphorin 3A causes immune suppression by inducing cytoskeletal paralysis in tumour-specific CD8+ T cells
Publisher Correction: Semaphorin 3A causes immune suppression by inducing cytoskeletal paralysis in tumour-specific CD8+ T cells
Movie S4 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Movie S3 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Supplemental File For Publication from Enriched HLA-E and CD94/NKG2A Interaction Limits Antitumor CD8<sup>+</sup> Tumor-Infiltrating T Lymphocyte Responses
Movie S1 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Movie S1 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Data from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Supplementary Movie Titles and Legends from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Data from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Movie S2 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Data from Enriched HLA-E and CD94/NKG2A Interaction Limits Antitumor CD8<sup>+</sup> Tumor-Infiltrating T Lymphocyte Responses
Data from Enhanced Immunogenicity of Mitochondrial-Localized Proteins in Cancer Cells
Supplementary File S1 from Enhanced Immunogenicity of Mitochondrial-Localized Proteins in Cancer Cells
Supplementary Figures and Tables from Enhanced Immunogenicity of Mitochondrial-Localized Proteins in Cancer Cells
Supplementary Fig S1-7, Sup Table 1 from Enriched HLA-E and CD94/NKG2A Interaction Limits Antitumor CD8<sup>+</sup> Tumor-Infiltrating T Lymphocyte Responses
Supplementary Fig S1-7, Sup Table 1 from Enriched HLA-E and CD94/NKG2A Interaction Limits Antitumor CD8<sup>+</sup> Tumor-Infiltrating T Lymphocyte Responses
Data from Enriched HLA-E and CD94/NKG2A Interaction Limits Antitumor CD8<sup>+</sup> Tumor-Infiltrating T Lymphocyte Responses
Supplementary Figures 1-7 and Supplementary Tables 1-2 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Supplementary File S1 from Enhanced Immunogenicity of Mitochondrial-Localized Proteins in Cancer Cells
Data from Enhanced Immunogenicity of Mitochondrial-Localized Proteins in Cancer Cells
Supplemental File For Publication from Enriched HLA-E and CD94/NKG2A Interaction Limits Antitumor CD8<sup>+</sup> Tumor-Infiltrating T Lymphocyte Responses
Supplementary Figures 1-7 and Supplementary Tables 1-2 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Supplementary Figures and Tables from Enhanced Immunogenicity of Mitochondrial-Localized Proteins in Cancer Cells
Movie S4 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Movie S3 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Supplementary Movie Titles and Legends from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
Movie S2 from Self-Maintaining CD103<sup>+</sup> Cancer-Specific T Cells Are Highly Energetic with Rapid Cytotoxic and Effector Responses
The P5-type ATPase ATP13A1 modulates major histocompatibility complex I-related protein 1 (MR1)-mediated antigen presentation.
Chromatin accessibility governs the differential response of cancer and T cells to arginine starvation