Area of research
Molecular Biology · Cancer Research
Research interest
Research interests include RNA modifications and cancer, Histone Deacetylase Inhibitors Research, Circular RNAs in diseases, and Cancer, Hypoxia, and Metabolism.
AARS2-catalyzed lactylation induces follicle development and premature ovarian insufficiency
N-homocysteinylation of β-arrestins biases GPCR signaling and promotes platelet activation
AARS2-catalyzed lactylation induces follicle development and premature ovarian insufficiency.
PPTC7 acts as an essential co-factor of the SCF<sup>FBXL4</sup> ubiquitin ligase complex to restrict BNIP3/3L-dependent mitophagy.
Metabolite is a part of immune-regulating circuit.
Correction: Calcineurin inactivation inhibits pyruvate dehydrogenase complex activity and induces the Warburg effect.
Hypoxia induces mitochondrial protein lactylation to limit oxidative phosphorylation
The alanyl-tRNA synthetase AARS1 moonlights as a lactyltransferase to promote YAP signaling in gastric cancer
Hypoxia induces mitochondrial protein lactylation to limit oxidative phosphorylation.
Ketogenic diet-produced β-hydroxybutyric acid accumulates brain GABA and increases GABA/glutamate ratio to inhibit epilepsy
Follicle stimulating hormone controls granulosa cell glutamine synthesis to regulate ovulation
DDHD2, whose mutations cause spastic paraplegia type 54, enhances lipophagy via engaging ATG8 family proteins
DDHD2, whose mutations cause spastic paraplegia type 54, enhances lipophagy via engaging ATG8 family proteins.
OTU deubiquitinase, ubiquitin aldehyde binding 2 (OTUB2) modulates the stemness feature, chemoresistance, and epithelial-mesenchymal transition of colon cancer via regulating GINS complex subunit 1 (GINS1) expression
SULF1 regulates malignant progression of colorectal cancer by modulating ARSH via FAK/PI3K/AKT/mTOR signaling
Tryptophanylation of insulin receptor by WARS attenuates insulin signaling
FBXL4 mutations cause excessive mitophagy via BNIP3/BNIP3L accumulation leading to mitochondrial DNA depletion syndrome
FBXL4 mutations cause excessive mitophagy via BNIP3/BNIP3L accumulation leading to mitochondrial DNA depletion syndrome.
Amino acids downregulate SIRT4 to detoxify ammonia through the urea cycle
m6A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR.
Amino acids downregulate SIRT4 to detoxify ammonia through the urea cycle.
The roles of NOP56 in cancer and SCA36
Data from m<sup>6</sup>A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR
Table S4 from m<sup>6</sup>A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR
Figure S3 from m<sup>6</sup>A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR
Figure S1 from m<sup>6</sup>A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR
Figure S5 from m<sup>6</sup>A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR
Table S2 from m<sup>6</sup>A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR
Table S3 from m<sup>6</sup>A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR
Table S1 from m<sup>6</sup>A-Mediated Biogenesis of circDDIT4 Inhibits Prostate Cancer Progression by Sequestrating ELAVL1/HuR