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Boaz Barak

Massachusetts Institute of Technology · US
Area of research
Developmental Neuroscience · Genetics
Research interest
Research interests include Biology, Neuroscience, Computational biology, RNA, Computer science, and Single-cell analysis.
h-index
citations
2,245
works
14
NIH funding
primary concept
email

Recent publications

HLA export by melanoma cells decoys cytotoxic T cells to promote immune evasion
Cell 2025cited by 6position: middledoi
Gtf2i-encoded transcription factor Tfii-i regulates myelination via Sox10 and Mbp regulatory elements
Nature Communications 2025cited by 1position: lastdoi
Neuronal Gtf2i deletion alters mitochondrial and autophagic properties
Communications Biology 2023cited by 7position: lastdoi
In individuals with Williams syndrome, dysregulation of methylation in non-coding regions of neuronal and oligodendrocyte DNA is associated with pathology and cortical development
Molecular Psychiatry 2022cited by 8position: lastdoi
Williams syndrome
Nature Reviews Disease Primers 2021cited by 240position: middledoi
MyelTracer: A Semi-Automated Software for Myelin<i>g</i>-Ratio Quantification
eNeuro 2021cited by 105position: middledoi
Systematic comparison of single-cell and single-nucleus RNA-sequencing methods
Nature Biotechnology 2020cited by 953position: middledoi
An Ultra-Sensitive Step-Function Opsin for Minimally Invasive Optogenetic Stimulation in Mice and Macaques
Neuron 2020cited by 159position: middledoi
Author Correction: Systematic comparison of single-cell and single-nucleus RNA-sequencing methods
Nature Biotechnology 2020cited by 16position: middledoi
Shank3 mutation in a mouse model of autism leads to changes in the S-nitroso-proteome and affects key proteins involved in vesicle release and synaptic function
Molecular Psychiatry 2018cited by 98position: middledoi
Direct modulation of GFAP-expressing glia in the arcuate nucleus bi-directionally regulates feeding
eLife 2016cited by 116position: middledoi
Mice with Shank3 Mutations Associated with ASD and Schizophrenia Display Both Shared and Distinct Defects
Neuron 2015cited by 332position: middledoi
Toll-like receptors as developmental tools that regulate neurogenesis during development: an update
DOAJ (DOAJ: Directory of Open Access Journals) 2014cited by 79position: firstdoi
Evidence for a Developmental Role for TLR4 in Learning and Memory
PLoS ONE 2012cited by 125position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Guoping Feng · Broad Institute5 papers (2015–2021)Tobias Kaiser · Massachusetts Institute of Technology3 papers (2015–2021)Tali Rosenberg · Hebrew University of Jerusalem3 papers (2022–2025)Zhanyan Fu · Massachusetts Institute of Technology3 papers (2015–2020) · 3 papers (2022–2025) · 3 papers (2022–2025)Hadar Parnas · Hebrew University of Jerusalem3 papers (2022–2025) · 3 papers (2022–2025)Asaf Marco · Hebrew University of Jerusalem3 papers (2022–2025)Nemanja D. Marjanovic · Broad Institute2 papers (2020–2020)William Ge · Dana-Farber Cancer Institute2 papers (2020–2020) · 2 papers (2020–2020)Aviv Regev · Moscow Institute of Thermal Technology2 papers (2020–2020)Marc H. Wadsworth · The Fertilizer Institute2 papers (2020–2020) · 2 papers (2023–2025)Sean Simmons · Massachusetts Institute of Technology2 papers (2020–2020)John Kwon · Weill Cornell Medicine2 papers (2020–2020)Lan Nguyễn · Broad Institute2 papers (2020–2020)Amanda J. Kedaigle · Massachusetts Institute of Technology2 papers (2020–2020)Joshua Z. Levin · Massachusetts Institute of Technology2 papers (2020–2020)