Area of research
Molecular Biology · Physiology
Research interest
Research interests include Biology, Cachexia, Skeletal muscle, Transcriptome, Medicine, and Atrophy.
At the Nexus Between Epigenetics and Senescence: The Effects of Senolytic ( <scp>BI01</scp> ) Administration on <scp>DNA</scp> Methylation Clock Age and the Methylome in Aged and Regenerated Skeletal Muscle
Sustained Accumulation of Molecular Clock Suppressors Period 1 and Period 2 Promotes C2C12 Myotube Atrophy Through an Autocrine-Mediated Mechanism With Relevance to Androgen Deprivation-Induced Limb Muscle Mass Loss
Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia
Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
The mechanosensitive gene <i>arrestin domain containing 2</i> regulates myotube diameter with direct implications for disuse atrophy with aging
Muscle weakness and mitochondrial stress occur before metastasis in a novel mouse model of ovarian cancer cachexia
Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
Multi-transcriptome analysis following an acute skeletal muscle growth stimulus yields tools for discerning global and MYC regulatory networks
A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
Muscle weakness precedes atrophy during cancer cachexia and is associated with muscle-specific mitochondrial stress
Muscle alterations in the development and progression of cancer-induced muscle atrophy: a review
A PGC-1α Isoform Induced by Resistance Training Regulates Skeletal Muscle Hypertrophy