Area of research
Molecular Biology · Pathology and Forensic Medicine
Research interest
Research interests include RNA modifications and cancer, Epigenetics and DNA Methylation, Genetic factors in colorectal cancer, and Genetic Associations and Epidemiology.
Genetic variations interact with polybrominated diphenyl ether exposure to alter lipid homeostasis.
Per- and Polyfluoroalkyl Substances Compromise Maternal Immune Protection via Suppressed Secretory IgA in Human Milk: Evidence from Population and Mechanistic Studies
Comprehensive analysis of transplacental transfer of environmental pollutants detected in paired maternal and cord serums
Single-cell eQTL Mapping Reveals Cell Subtype-specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer.
Genetic architecture of bone marrow fat fraction implies its involvement in osteoporosis risk.
Genetic Control of tRNA-Derived Fragments Contributes to Cancer Risk.
Genetic profiling of the circulating proteome in common diseases suggests causal proteins and improves risk prediction.
Characterization of cis-regulatory elements and functional variants in colorectal cancer using epigenomics and CRISPRi screenings.
Author Correction: Exome-wide analysis identifies three low-frequency missense variants associated with pancreatic cancer risk in Chinese populations.
Supplementary Figure S34 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S4 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S25 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S10 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Data from Genetic Control of tRNA-Derived Fragments Contributes to Cancer Risk
Figure S6 from Genetic Control of tRNA-Derived Fragments Contributes to Cancer Risk
Supplementary Figure S7 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S37 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S30 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S33 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S5 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S6 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S19 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Tables S1-S17 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Methods from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Figure S8 from Genetic Control of tRNA-Derived Fragments Contributes to Cancer Risk
Supplementary Figure S23 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Figure S15 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Figure S10 from Genetic Control of tRNA-Derived Fragments Contributes to Cancer Risk
Supplementary Figure S28 from Single-cell eQTL Mapping Reveals Cell Subtype–specific Genetic Control and Mechanism in Malignant Transformation of Colorectal Cancer
Supplementary Data from Genetic Control of tRNA-Derived Fragments Contributes to Cancer Risk