Area of research
Pulmonary and Respiratory Medicine · Oncology
Research interest
Research interests include Pancreatic and Hepatic Oncology Research, Kidney Stones and Urolithiasis Treatments, Gallbladder and Bile Duct Disorders, and Epigenetics and DNA Methylation.
HNF1B-driven three-dimensional chromatin structure for molecular classification in pancreatic cancers.
Autophagy as a critical driver of metabolic adaptation, therapeutic resistance, and immune evasion of cancer.
Targeting the Metabolic Rewiring in Pancreatic Cancer and Its Tumor Microenvironment.
Targeting autophagy as a therapeutic strategy against pancreatic cancer.
MNX1-HNF1B Axis Is Indispensable for Intraductal Papillary Mucinous Neoplasm Lineages.
Autophagy is required for proper cysteine homeostasis in pancreatic cancer through regulation of SLC7A11
Autophagy is required for proper cysteine homeostasis in pancreatic cancer through regulation of SLC7A11.
Functional Genomics Identifies Metabolic Vulnerabilities in Pancreatic Cancer.
The polar oxy-metabolome reveals the 4-hydroxymandelate CoQ10 synthesis pathway.
Inhibition of histone methyltransferase G9a attenuates liver cancer initiation by sensitizing DNA-damaged hepatocytes to p53-induced apoptosis.
Autophagy promotes immune evasion of pancreatic cancer by degrading MHC-I
Autophagy promotes immune evasion of pancreatic cancer by degrading MHC-I.
Neurons Release Serine to Support mRNA Translation in Pancreatic Cancer
Neurons Release Serine to Support mRNA Translation in Pancreatic Cancer.
Selective Alanine Transporter Utilization Creates a Targetable Metabolic Niche in Pancreatic Cancer
Selective Alanine Transporter Utilization Creates a Targetable Metabolic Niche in Pancreatic Cancer.
Selective autophagy of MHC-I promotes immune evasion of pancreatic cancer
Functional Genomics Identifies Metabolic Vulnerabilities in Pancreatic Cancer
Selective autophagy of MHC-I promotes immune evasion of pancreatic cancer.
A role for gorilla APOBEC3G in shaping lentivirus evolution including transmission to humans
Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancreatic cancer.
5-Aminolevulinic acid-mediated photodynamic activity in patient-derived cholangiocarcinoma organoids.
Deletion of Histone Methyltransferase G9a Suppresses Mutant Kras-driven Pancreatic Carcinogenesis.
Mutant IDH1 confers resistance to energy stress in normal biliary cells through PFKP-induced aerobic glycolysis and AMPK activation.