Area of research
Neurology · Oncology
Research interest
Research interests include Neuroinflammation and Neurodegeneration Mechanisms, Traumatic Brain Injury and Neurovascular Disturbances, Cancer Treatment and Pharmacology, and Stress Responses and Cortisol.
Fate mapping of peripherally-derived macrophages after traumatic brain injury in mice reveals a long-lasting population with a distinct transcriptomic signature
Anti-acetylated-tau immunotherapy is neuroprotective in tauopathy and brain injury
Novel microglia-mediated mechanisms underlying synaptic loss and cognitive impairment after traumatic brain injury
The impact of deep space radiation on cognitive performance: From biological sex to biomarkers to countermeasures
Small molecule cognitive enhancer reverses age-related memory decline in mice
Small molecule cognitive enhancer reverses age-related memory decline in mice
Integrated Stress Response Inhibitor Reverses Sex-Dependent Behavioral and Cell-Specific Deficits after Mild Repetitive Head Trauma
Traumatic Brain Injury in Aged Mice Induces Chronic Microglia Activation, Synapse Loss, and Complement-Dependent Memory Deficits
Female mice are protected from space radiation-induced maladaptive responses
Temporary microglia-depletion after cosmic radiation modifies phagocytic activity and prevents cognitive deficits
Persistent Infiltration and Impaired Response of Peripherally-Derived Monocytes after Traumatic Brain Injury in the Aged Brain
Repeated Mild Head Injury Leads to Wide-Ranging Deficits in Higher-Order Cognitive Functions Associated with the Prefrontal Cortex
Inhibition of the integrated stress response reverses cognitive deficits after traumatic brain injury
In vivo metabolic imaging of Traumatic Brain Injury
CD8<sup>+</sup>T Cells and Endogenous IL-10 Are Required for Resolution of Chemotherapy-Induced Neuropathic Pain
The Anti-Diabetic Drug Metformin Protects against Chemotherapy-Induced Peripheral Neuropathy in a Mouse Model
Glucocorticoids regulate natural killer cell function epigenetically
Glucocorticoid receptor mediated suppression of natural killer cell activity: Identification of associated deacetylase and corepressor molecules