Ineke Braakman

The Folding Pathway of ABC Transporter CFTR: Effective and Robust

ABC-transporter CFTR folds with high fidelity through a modular, stepwise pathway

Redefining Hypo- and Hyper-Responding Phenotypes of CFTR Mutants for Understanding and Therapy

Co-Translational Folding of the First Transmembrane Domain of ABC-Transporter CFTR is Supported by Assembly with the First Cytosolic Domain

The CFTR P67L variant reveals a key role for N-terminal lasso helices in channel folding, maturation, and pharmacologic rescue

<scp>The importance of naturally attenuated SARS‐CoV</scp> ‐2 <scp>in the fight against COVID</scp> ‐19

Slowing ribosome velocity restores folding and function of mutant CFTR

Folding–function relationship of the most common cystic fibrosis–causing CFTR conductance mutants

Characterization of CNPY5 and its family members

CHAPTER 1.1. Disulfide Bonds in Protein Folding and Stability

Alteration of protein function by a silent polymorphism linked to tRNA abundance

Correcting CFTR folding defects by small-molecule correctors to cure cystic fibrosis

Structure and topology around the cleavage site regulate post-translational cleavage of the HIV-1 gp160 signal peptide

Co‐ and Post‐Translational Protein Folding in the <scp>ER</scp>

Protein quality control at the endoplasmic reticulum

TORC2 mediates the heat stress response in <i>Drosophila</i> by promoting the formation of stress granules

Deletion of the Highly Conserved N-Glycan at Asn260 of HIV-1 gp120 Affects Folding and Lysosomal Degradation of gp120, and Results in Loss of Viral Infectivity

Two phases of disulfide bond formation have differing requirements for oxygen

ERdj5 Is the ER Reductase that Catalyzes the Removal of Non-Native Disulfides and Correct Folding of the LDL Receptor

Quantifying Changes in the Cellular Thiol-Disulfide Status during Differentiation of B Cells into Antibody-Secreting Plasma Cells

Peroxisome Formation and Maintenance Are Dependent on the Endoplasmic Reticulum

Ero1–PDI interactions, the response to redox flux and the implications for disulfide bond formation in the mammalian endoplasmic reticulum

Biochemically Distinct Vesicles from the Endoplasmic Reticulum Fuse to Form Peroxisomes