Daniela Kramer

Regulation and Function of the Atypical IκBs—Bcl‐3, IκB _NS , and IκBζ—in Lymphocytes and Autoimmunity

NOD2-Induced IκBζ Mediates a Protective Host Response against Epicutaneous Staphylococcus aureus Infection

naRNA-LL37 composite DAMPs define sterile NETs as self-propagating drivers of inflammation

LL37/self-DNA complexes mediate monocyte reprogramming

Epidermal proteomics demonstrates Elafin as a psoriasis‐specific biomarker and highlights increased anti‐inflammatory activity around psoriatic plaques

The atypical IκB family member Bcl3 determines differentiation and fate of intestinal RORγt+ regulatory T-cell subsets

Acidosis-mediated increase in IFN-γ-induced PD-L1 expression on cancer cells as an immune escape mechanism in solid tumors

IL-17A-driven psoriasis is critically dependent on IL-36 signaling

Immunomodulatory Effects of Histone Variant H2A.J in Ionizing Radiation Dermatitis

The CDK4/6-EZH2 pathway is a potential therapeutic target for psoriasis

Immunomodulatory role of reactive oxygen species and nitrogen species during T cell-driven neutrophil-enriched acute and chronic cutaneous delayed-type hypersensitivity reactions

Threonine Phosphorylation of IκBζ Mediates Inhibition of Selective Proinflammatory Target Genes

Keratinocyte-derived IκBζ drives psoriasis and associated systemic inflammation

Targeting chronic NFAT activation with calcineurin inhibitors in diffuse large B-cell lymphoma

Temporal Dynamics of Reactive Oxygen and Nitrogen Species and NF-κB Activation During Acute and Chronic T Cell–Driven Inflammation

IκBζ is a key transcriptional regulator of IL-36–driven psoriasis-related gene expression in keratinocytes

Subcutaneous immunoglobulin for maintenance treatment in chronic inflammatory demyelinating polyneuropathy (PATH): a randomised, double-blind, placebo-controlled, phase 3 trial