Area of research
Genetics · Cancer Research
Research interest
Research interests include Glioma Diagnosis and Treatment, Epigenetics and DNA Methylation, Brain Metastases and Treatment, and Cancer Genomics and Diagnostics.
cIMPACT-NOW update 11: Proposal on adaptation of diagnostic criteria for IDH- and H3-wildtype diffuse high-grade gliomas and for posterior fossa ependymal tumors.
Developing an advanced risk stratification model for pediatric intracranial ependymoma based on the prospective trial E-HIT2000 and subsequent registries.
Functional synapses between neurons and small cell lung cancer
Advancing CNS tumor diagnostics with expanded DNA methylation-based classification
<scp>cIMPACT</scp> ‐ <scp>NOW</scp> update 11: Proposal on adaptation of diagnostic criteria for <scp>IDH</scp> ‐ and <scp>H3</scp> ‐wildtype diffuse high‐grade gliomas and for posterior fossa ependymal tumors
The clinical and molecular landscape of diffuse hemispheric glioma, H3 G34-mutant
Strong nuclear expression of HOXB13 is a reliable surrogate marker for DNA methylome profiling to distinguish myxopapillary ependymoma from spinal ependymoma
The clinical and molecular landscape of diffuse hemispheric glioma, H3 G34-mutant.
<scp>cIMPACT</scp>‐<scp>NOW</scp> update 10: Recommendations for defining new types for central nervous system tumor classification
IDH-mutant astrocytomas with primitive neuronal component have a distinct methylation profile and a higher risk of leptomeningeal spread
Advancing CNS tumor diagnostics with expanded DNA methylation-based classification
Developing an advanced risk stratification model for pediatric intracranial ependymoma based on the prospective trial E-HIT2000 and subsequent registries
Loss of global DNA hypermethylation is prognostic in IDH-mutant and 1p/19q-codeleted oligodendrogliomas
Strong nuclear expression of HOXB13 is a reliable surrogate marker for DNA methylome profiling to distinguish myxopapillary ependymoma from spinal ependymoma
Publisher Correction: Functional synapses between neurons and small cell lung cancer
Data from A Sequential Targeting Strategy Interrupts AKT-Driven Subclone-Mediated Progression in Glioblastoma
Supplementary Figure S3 from A Sequential Targeting Strategy Interrupts AKT-Driven Subclone-Mediated Progression in Glioblastoma
Supplementary Tables S1-4 from A Sequential Targeting Strategy Interrupts AKT-Driven Subclone-Mediated Progression in Glioblastoma
Supplementary Figure S2 from A Sequential Targeting Strategy Interrupts AKT-Driven Subclone-Mediated Progression in Glioblastoma
Supplementary Figure S4 from A Sequential Targeting Strategy Interrupts AKT-Driven Subclone-Mediated Progression in Glioblastoma
Supplementary Figure S1 from A Sequential Targeting Strategy Interrupts AKT-Driven Subclone-Mediated Progression in Glioblastoma
The oncological role of resection in newly diagnosed diffuse adult-type glioma defined by the WHO 2021 classification: a Review by the RANO resect group
The biological significance of tumor grade, age, enhancement, and extent of resection in IDH-mutant gliomas: How should they inform treatment decisions in the era of IDH inhibitors?
The biological significance of tumor grade, age, enhancement, and extent of resection in IDH-mutant gliomas: How should they inform treatment decisions in the era of IDH inhibitors?
Improved prognostic stratification of patients with isocitrate dehydrogenase-mutant astrocytoma
The Epigenetic Evolution of Glioma Is Determined by the IDH1 Mutation Status and Treatment Regimen.
mTORC1 regulates cell survival under glucose starvation through 4EBP1/2-mediated translational reprogramming of fatty acid metabolism
European Association of Neuro-Oncology guideline on molecular testing of meningiomas for targeted therapy selection
Direct excitatory synapses between neurons and tumor cells drive brain metastatic seeding of breast cancer and melanoma