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Nathan K. Archer

Johns Hopkins Medicine · US
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Area of research
Dermatology · Immunology
Research interest
Research interests include Immunology, Biology, Inflammation, Staphylococcus aureus, Medicine, and Psoriasis.
h-index
citations
1,200
works
13
NIH funding
primary concept
email

Recent publications

naRNA-LL37 composite DAMPs define sterile NETs as self-propagating drivers of inflammation
EMBO Reports 2024cited by 23position: middledoi
Keratinocyte-derived defensins activate neutrophil-specific receptors Mrgpra2a/b to prevent skin dysbiosis and bacterial infection
Immunity 2022cited by 41position: middledoi
Staphylococcus aureus adaptive evolution: Recent insights on how immune evasion, immunometabolic subversion and host genetics impact vaccine development
Frontiers in Cellular and Infection Microbiology 2022cited by 39position: middledoi
Dendritic cell immunoreceptor drives atopic dermatitis by modulating oxidized CaMKII-involved mast cell activation
JCI Insight 2022cited by 34position: middledoi
Crisaborole efficacy in murine models of skin inflammation and <scp><i>Staphylococcus aureus</i></scp> infection
Experimental Dermatology 2022cited by 3position: lastdoi
Bacteria induce skin regeneration via IL-1β signaling
Cell Host & Microbe 2021cited by 196position: middledoi
Tick extracellular vesicles enable arthropod feeding and promote distinct outcomes of bacterial infection
Nature Communications 2021cited by 61position: middledoi
Dynamic PET-facilitated modeling and high-dose rifampin regimens for <i>Staphylococcus aureus</i> orthopedic implant–associated infections
Science Translational Medicine 2021cited by 28position: middledoi
Neutrophil extracellular trap-associated RNA and LL37 enable self-amplifying inflammation in psoriasis
Nature Communications 2020cited by 269position: middledoi
Clonal Vγ6 <sup>+</sup> Vδ4 <sup>+</sup> T cells promote IL-17–mediated immunity against <i>Staphylococcus aureus</i> skin infection
Proceedings of the National Academy of Sciences 2019cited by 109position: middledoi
Platelets Aggregate With Neutrophils and Promote Skin Pathology in Psoriasis
Frontiers in Immunology 2019cited by 44position: middledoi
Clonally expanded γδ T cells protect against Staphylococcus aureus skin reinfection
Journal of Clinical Investigation 2018cited by 102position: middledoi
Staphylococcus aureus Epicutaneous Exposure Drives Skin Inflammation via IL-36-Mediated T Cell Responses
Cell Host & Microbe 2017cited by 251position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Lloyd Miller · Johnson & Johnson (United States)9 papers (2017–2022)Martin P. Alphonse · Hospital for Sick Children5 papers (2019–2022)Yu Wang · Anhui Medical University5 papers (2017–2022)Roger V. Ortines · Johns Hopkins Medicine4 papers (2017–2022)Haiyun Liu · China Pharmaceutical University4 papers (2017–2022)Michael R. Yeaman · LAC+USC Medical Center3 papers (2018–2022)Alexander N.R. Weber · German Cancer Research Center3 papers (2019–2024)Markus Löffler · Research Institute for Work, Technology and Culture3 papers (2019–2024)Christine Youn · AstraZeneca (Singapore)3 papers (2022–2024)Xinzhong Dong · Howard Hughes Medical Institute3 papers (2019–2022)Shuting Cai · Columbia University3 papers (2017–2019)Carly Dillen · Johns Hopkins Medicine3 papers (2017–2019)Mark Marchitto · Johns Hopkins Medicine3 papers (2017–2019)Dustin Dikeman · Johns Hopkins Medicine3 papers (2021–2022)Scott I. Simon · University of California, Davis2 papers (2018–2019)Emanual Maverakis · University of California Davis Medical Center2 papers (2018–2019)Alexander A. Merleev · University of California, Davis2 papers (2018–2019)Luis A. Garza · Johns Hopkins Medicine2 papers (2021–2022)Kamran Ghoreschi · Clinical Research Center for Hair and Skin Science2 papers (2019–2020)Zsofia Bittner · University of Tübingen2 papers (2019–2020)
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