Karin Lykke‐Hartmann

Abnormal neurovascular coupling as a cause of excess cerebral vasodilation in familial migraine

The α2β2 isoform combination dominates the astrocytic Na⁺/K⁺-ATPase activity and is rendered nonfunctional by the α2.G301R familial hemiplegic migraine type 2-associated mutation

Glutamate-system defects behind psychiatric manifestations in a familial hemiplegic migraine type 2 disease-mutation mouse model