Research interests include IL-33, ST2, and ILC Pathways, Psoriasis: Treatment and Pathogenesis, Eosinophilic Esophagitis, and Cholesterol and Lipid Metabolism.
Distinct expression of interleukin (IL)-36<b>α, β</b> and <b>γ</b>, their antagonist IL-36Ra and IL-38 in psoriasis, rheumatoid arthritis and Crohn's disease
Clinical & Experimental Immunology2015cited by 266position: middledoi
IL-36 signaling amplifies Th1 responses by enhancing proliferation and Th1 polarization of naive CD4+ T cells