Area of research
Cardiology and Cardiovascular Medicine · Endocrinology, Diabetes and Metabolism
Research interest
Research interests include Renin-Angiotensin System Studies, Hormonal Regulation and Hypertension, Ion Transport and Channel Regulation, and Nitric Oxide and Endothelin Effects.
Loss of Soluble (Pro)renin Receptor Attenuates Angiotensin-II Induced Hypertension and Renal Injury
Collecting duct principal, but not intercalated, cell prorenin receptor regulates renal sodium and water excretion
Renal tubular epithelial cell prorenin receptor regulates blood pressure and sodium transport
Nephron-specific deletion of the prorenin receptor causes a urine concentration defect
Collecting duct-specific knockout of renin attenuates angiotensin II-induced hypertension
Smooth muscle specific disruption of the endothelin-A receptor in mice reduces arterial pressure, and vascular reactivity and affects vascular development
Overexpression of Renin in the Collecting Duct Causes Elevated Blood Pressure
Myocardial, Smooth Muscle, Nephron, and Collecting Duct Gene Targeting Reveals the Organ Sites of Endothelin A Receptor Antagonist Fluid Retention
A Possible Interaction Between Systemic and Renal Angiotensinogen in the Control of Blood Pressure
Overexpression of mouse angiotensinogen in renal proximal tubule causes salt-sensitive hypertension in mice
Disruption of the endothelin A receptor in the nephron causes mild fluid volume expansion