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Hiroshi Ueda

Community Research Initiative · JP
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Area of research
Cellular and Molecular Neuroscience · Physiology
Research interest
Research interests include Neuropeptides and Animal Physiology, Pain Mechanisms and Treatments, Receptor Mechanisms and Signaling, and Neuroscience and Neuropharmacology Research.
h-index
60
citations
14,691
works
803
NIH funding
primary concept
email

Recent publications

A novel lysophosphatidic acid receptor 1 antagonist with high brain penetrability has a curative effect in the empathic pain-related fibromyalgia model.
2025cited by 0position: contributordoi
Single-molecule detection of transient dimerization of opioid receptors 2: Heterodimer blockage reduces morphine tolerance
2024cited by 1position: contributordoi
Single-molecule detection of transient dimerization of opioid receptors 2: Heterodimer blockage reduces morphine tolerance
2024cited by 0position: contributordoi
Fibromyalgia Animal Models Using Intermittent Cold and Psychological Stress.
2023cited by 5position: contributordoi
Prothymosin α-derived hexapeptide prevents the brain damage and sequelae due to ischemia-hemorrhage.
2023cited by 1position: contributordoi
Lysophosphatidic acid receptor type-1 mediates brain activation in micro-positron emission tomography analysis in a fibromyalgia-like mouse model.
2022cited by 1position: contributordoi
Pathogenic mechanisms of lipid mediator lysophosphatidic acid in chronic pain.
2021cited by 29position: contributordoi
Secreted PLA<sub>2</sub>-III is a possible therapeutic target to treat neuropathic pain.
2021cited by 8position: contributordoi
Annexin A2 Flop-Out Mediates the Non-Vesicular Release of DAMPs/Alarmins from C6 Glioma Cells Induced by Serum-Free Conditions.
2021cited by 3position: contributordoi
Involvement of SNARE Protein Interaction for Non-classical Release of DAMPs/Alarmins Proteins, Prothymosin Alpha and S100A13.
2021cited by 2position: contributordoi
LPA receptor signaling as a therapeutic target for radical treatment of neuropathic pain and fibromyalgia.
2020cited by 24position: contributordoi
NR2A-NMDA Receptor Blockade Reverses the Lack of Morphine Analgesia Without Affecting Chronic Pain Status in a Fibromyalgia-Like Mouse Model.
2020cited by 13position: contributordoi
Prothymosin alpha and its mimetic hexapeptide improve delayed tissue plasminogen activator-induced brain damage following cerebral ischemia.
2020cited by 9position: contributordoi
Lysophosphatidic Acid Receptor 1- and 3-Mediated Hyperalgesia and Hypoalgesia in Diabetic Neuropathic Pain Models in Mice.
2020cited by 8position: contributordoi
Allodynia by Splenocytes From Mice With Acid-Induced Fibromyalgia-Like Generalized Pain and Its Sexual Dimorphic Regulation by Brain Microglia.
2020cited by 7position: contributordoi
Gγ7-specific prothymosin alpha deletion causes stress- and age-dependent motor dysfunction and anxiety.
2020cited by 6position: contributordoi
Experimental evidence for the involvement of F<sub>0</sub>/F<sub>1</sub> ATPase and subsequent P2Y<sub>12</sub> receptor activation in prothymosin alpha-induced protection of retinal ischemic damage.
2020cited by 6position: contributordoi
Lysophosphatidic acid LPA<sub>1</sub> and LPA<sub>3</sub> receptors play roles in the maintenance of late tissue plasminogen activator-induced central poststroke pain in mice.
2019cited by 32position: contributordoi
Regulation of an Opioid Receptor Chaperone Protein, RTP4, by Morphine.
2019cited by 18position: contributordoi
Critical Functionality Effects from Storage Temperature on Human Induced Pluripotent Stem Cell-Derived Retinal Pigment Epithelium Cell Suspensions.
2019cited by 18position: contributordoi
Systems Pathology of Neuropathic Pain and Fibromyalgia.
2019cited by 12position: contributordoi
Abrogation of lysophosphatidic acid receptor 1 ameliorates murine vasculitis.
2019cited by 11position: contributordoi
Opioid receptors (version 2019.4) in the IUPHAR/BPS Guide to Pharmacology Database
IUPHAR/BPS Guide to Pharmacology CITE 2019cited by 11position: middledoi
[Lysophosphatidic Acid Receptor Signaling Underlying Chronic Pain and Neuroprotective Mechanisms through Prothymosin α].
2019cited by 0position: contributordoi

Grants

No grants ingested yet.

Frequent collaborators

· 11 papers (2020–2025)Hiroyuki Neyama · Kyoto University4 papers (2020–2023)Shuji Kaneko · Kyushu University2 papers (2020–2021)Takahiro K. Fujiwara · Saitama University2 papers (2024–2024)Wakako Fujita · Kobe University2 papers (2024–2024)Peng Zhou · Kyoto University2 papers (2024–2024)Hayato Matsunaga · Nagasaki University2 papers (2021–2021)Simone Pigolotti · Okinawa Institute of Science and Technology Graduate University2 papers (2024–2024)Rinshi S. Kasai · Kyoto University2 papers (2024–2024)Akihiro Kusumi · Okinawa Institute of Science and Technology Graduate University2 papers (2024–2024)Taka A. Tsunoyama · Okinawa Institute of Science and Technology Graduate University2 papers (2024–2024)Sebok Kumar Halder · Nagasaki University1 papers (2021–2021)Kazuki Nagayasu · American Chemical Society1 papers (2021–2021)Yasuyoshi Watanabe · Kyoto University1 papers (2022–2022)
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