Area of research
Cellular and Molecular Neuroscience · Molecular Biology
Research interest
Research interests include Neuroscience and Neuropharmacology Research, Neurotransmitter Receptor Influence on Behavior, Receptor Mechanisms and Signaling, and Memory and Neural Mechanisms.
A ventral pallidal glutamatergic aversive network encodes abstinence from and reexposure to cocaine
Multimodal interrogation of ventral pallidum projections reveals projection-specific signatures and effects on cocaine reward
Cocaine induces input and cell-type-specific synaptic plasticity in ventral pallidum-projecting nucleus accumbens medium spiny neurons
Ventral pallidum cellular and pathway specificity in drug seeking
The role of the nucleus accumbens and ventral pallidum in feeding and obesity
Metaplasticity in the Ventral Pallidum as a Potential Marker for the Propensity to Gain Weight in Chronic High-Calorie Diet
Accumbens neuroimmune signaling and dysregulation of astrocytic glutamate transport underlie conditioned nicotine‐seeking behavior
Projection-Specific Potentiation of Ventral Pallidal Glutamatergic Outputs after Abstinence from Cocaine
Cocaine Dysregulates Dynorphin Modulation of Inhibitory Neurotransmission in the Ventral Pallidum in a Cell-Type-Specific Manner
Loss of Plasticity in the D2-Accumbens Pallidal Pathway Promotes Cocaine Seeking
The Nucleus Accumbens: Mechanisms of Addiction across Drug Classes Reflect the Importance of Glutamate Homeostasis
Loss of Plasticity in the D2-Accumbens Pallidal Pathway Promotes Cocaine Seeking
The Direct and Indirect Pathways of the Nucleus Accumbens are not What You Think
Coding the direct/indirect pathways by D1 and D2 receptors is not valid for accumbens projections
Optogenetic inhibition of cortical afferents in the nucleus accumbens simultaneously prevents cue-induced transient synaptic potentiation and cocaine-seeking behavior
Addiction-like Synaptic Impairments in Diet-Induced Obesity
Glutamatergic mechanisms of comorbidity between acute stress and cocaine self-administration
Optogenetic inhibition of cocaine seeking in rats