Area of research
Cell Biology · Molecular Biology
Research interest
Research interests include Unfolded protein response, Endoplasmic reticulum, Tunicamycin, Simvastatin, Wnt signaling pathway, and Signal transduction.
Unravelling the role of NFE2L1 in stress responses and related diseases
The Effect of Cholesterol Efflux on Endothelial Dysfunction Caused by Oxidative Stress
Endothelial Dysfunction Induced by Extracellular Neutrophil Traps Plays Important Role in the Occurrence and Treatment of Extracellular Neutrophil Traps-Related Disease
2-Hydroxypropyl-β-cyclodextrin Regulates the Epithelial to Mesenchymal Transition in Breast Cancer Cells by Modulating Cholesterol Homeostasis and Endoplasmic Reticulum Stress
Tunicamycin-Induced Endoplasmic Reticulum Stress Promotes Breast Cancer Cell MDA-MB-231 Apoptosis through Inhibiting Wnt/β-Catenin Signaling Pathway
Endoplasmic Reticulum Stress Affects Cholesterol Homeostasis by Inhibiting LXRα Expression in Hepatocytes and Macrophages
Sphingomyelin Synthase 2 Participate in the Regulation of Sperm Motility and Apoptosis
Individual factors define the overall effects of dietary genistein exposure on breast cancer patients
Sphingomyelin Synthase 2 Promotes Endothelial Dysfunction by Inducing Endoplasmic Reticulum Stress
Simvastatin Attenuates H2O2-Induced Endothelial Cell Dysfunction by Reducing Endoplasmic Reticulum Stress
Simvastatin promotes endothelial dysfunction by activating the Wnt/β‑catenin pathway under oxidative stress
Sphingomyelin synthase 2 promotes H2O2-induced endothelial dysfunction by activating the Wnt/β-catenin signaling pathway
Sphingomyelin synthase 1 regulates the epithelial‑to‑mesenchymal transition mediated by the TGF‑β/Smad pathway in MDA‑MB‑231 cells
Effects of sepsis on the metabolism of sphingomyelin and cholesterol in mice with liver dysfunction