Area of research
Pulmonary and Respiratory Medicine · Physiology
Research interest
Research interests include Nitric Oxide and Endothelin Effects, Pulmonary Hypertension Research and Treatments, Mitochondrial Function and Pathology, and Renin-Angiotensin System Studies.
c-Myc promotes metabolic reprogramming in pulmonary hypertension via the stimulation of glutaminolysis and the reductive tricarboxylic acid cycle
Mitochondrial network dynamics in pulmonary disease: Bridging the gap between inflammation, oxidative stress, and bioenergetics
Prognostic Value of a Classification System for Iliofemoral Stenting in Patients with Chronic Venous Obstruction
Metabolic reprogramming, oxidative stress, and pulmonary hypertension
SOX17 Deficiency Mediates Pulmonary Hypertension: At the Crossroads of Sex, Metabolism, and Genetics
Exploring m6A‐RNA methylation as a potential therapeutic strategy for acute lung injury and acute respiratory distress syndrome
Mitochondrial hyperfusion induces metabolic remodeling in lung endothelial cells by modifying the activities of electron transport chain complexes I and III
Nitration-mediated activation of the small GTPase RhoA stimulates cellular glycolysis through enhanced mitochondrial fission
PTK2-associated gene signature could predict the prognosis of IPF
The fatal trajectory of pulmonary COVID-19 is driven by lobular ischemia and fibrotic remodelling
Inflammation and vascular remodeling in COVID-19 hearts
The Novel Lysosomal Autophagy Inhibitor (ROC-325) Ameliorates Experimental Pulmonary Hypertension
Endothelial upregulation of mechanosensitive channel Piezo1 in pulmonary hypertension
The mitochondrial redistribution of eNOS is involved in lipopolysaccharide induced inflammasome activation during acute lung injury
Cytokines, Chemokines, and Inflammation in Pulmonary Arterial Hypertension
Complex interplay between autophagy and oxidative stress in the development of pulmonary disease
Tetramethylpyrazine: A promising drug for the treatment of pulmonary hypertension
Activation of the mechanosensitive Ca2+ channel TRPV4 induces endothelial barrier permeability via the disruption of mitochondrial bioenergetics
Advanced spray dried proliposomes of amphotericin B lung surfactant-mimic phospholipid microparticles/nanoparticles as dry powder inhalers for targeted pulmonary drug delivery
TGF-β1 attenuates mitochondrial bioenergetics in pulmonary arterial endothelial cells via the disruption of carnitine homeostasis
RAC1 nitration at Y32 IS involved in the endothelial barrier disruption associated with lipopolysaccharide-mediated acute lung injury
Inhalable Nanoparticles/Microparticles of an AMPK and Nrf2 Activator for Targeted Pulmonary Drug Delivery as Dry Powder Inhalers
Salusin-β Promotes Vascular Calcification <i>via</i> Nicotinamide Adenine Dinucleotide Phosphate/Reactive Oxygen Species-Mediated Klotho Downregulation
Biomechanical Forces and Oxidative Stress: Implications for Pulmonary Vascular Disease
Surfing the right ventricular pressure waveform: methods to assess global, systolic and diastolic RV function from a clinical right heart catheterization
Ovine Models of Congenital Heart Disease and the Consequences of Hemodynamic Alterations for Pulmonary Artery Remodeling
Pulmonary Endothelial Mechanical Sensing and Signaling, a Story of Focal Adhesions and Integrins in Ventilator Induced Lung Injury
Pathogenic Role of mTORC1 and mTORC2 in Pulmonary Hypertension
Divergent changes of p53 in pulmonary arterial endothelial and smooth muscle cells involved in the development of pulmonary hypertension
Mechanisms Behind Resistance to PI3K Inhibitor Treatment Induced by the PIM Kinase
Mito-inflammation and sepsis-induced acute lung injury
Development of Novel RhoA Nitration Inhibitory Peptides for the Treatment of Acute Lung Injury
FIU-Center for Genome Research (FIU-CGR).
Genetics, Epigenetics, and Post-translational Modifications and the Development of Ventilator Induced Lung Injury (VILI)
PKG Signaling and Sepsis Induced ALI
Metabolic Reprogramming and Pulmonary Vascular Disease in Congenital Heart Disease
METABOLIC REPROGRAMMING AND PULMONARY VASCULAR DISEASE ASSOCIATED WITH CONGENITAL HEART DISEASE RESULTING IN INCREASED PULMONARY BLOOD FLOW
Metabolic Reprogramming and Pulmonary Vascular Disease in Congenital Heart Disease
Post-translational modifications & mitochondrial bioenergetics
Mitochondrial Imaging, PTM Analysis and Metabolism Core
Protein Chemistry & Biophysics Core
Genetics, Epigenetics, and Post-translational Modifications and the Development of Ventilator-Induced Lung Injury (VILI)
PKG Signaling and Sepsis Induced ALI
PKG Signaling and Sepsis Induced ALI
PKG Signaling and Sepsis Induced ALI
Genetics, Epigenetics, and Post-translational Modifications and the Development of Ventilator-Induced Lung Injury (VILI)
Protein Chemistry & Biophysics Core
Perinatal NO signaling in congenital heart disease
Perinatal NO signaling in congenital heart disease
Perinatal NO signaling in congenital heart disease
Metabolic reprogramming of smooth muscle cells in pulmonary hypertension
Metabolic reprogramming of smooth muscle cells in pulmonary hypertension