Daniela Fraccarollo

Mineralocorticoid receptor promotes cardiac macrophage inflammaging

Circulating soluble fibroblast activation protein (FAP) levels are independent of cardiac and extra-cardiac FAP expression determined by targeted molecular imaging in patients with myocardial FAP activation

Expansion of CD10neg neutrophils and CD14+HLA-DRneg/low monocytes driving proinflammatory responses in patients with acute myocardial infarction

Gut Microbiota–Dependent Trimethylamine <i>N</i> -Oxide Predicts Risk of Cardiovascular Events in Patients With Stroke and Is Related to Proinflammatory Monocytes

Macrophage Mineralocorticoid Receptor Is a Pleiotropic Modulator of Myocardial Infarct Healing

The glucocorticoid receptor in monocyte‐derived macrophages is critical for cardiac infarct repair and remodeling

Mental disorders in adults with congenital heart disease: Unmet needs and impact on quality of life

C1q-TNF-Related Protein-9 Promotes Cardiac Hypertrophy and Failure

Multidrug resistance associated protein-1 (MRP1) deficiency attenuates endothelial dysfunction in diabetes

Fibroblast activation protein alpha expression identifies activated fibroblasts after myocardial infarction

Inflammatory Monocytes Determine Endothelial Nitric-oxide Synthase Uncoupling and Nitro-oxidative Stress Induced by Angiotensin II

Soluble guanylyl cyclase activation improves progressive cardiac remodeling and failure after myocardial infarction. Cardioprotection over ACE inhibition

The direct factor Xa inhibitor Rivaroxaban reduces platelet activation in congestive heart failure

Monocytes/macrophages prevent healing defects and left ventricular thrombus formation after myocardial infarction

Novel therapeutic approaches to post-infarction remodelling