Area of research
Sensory Systems · Physiology
Research interest
Research interests include Ion Channels and Receptors, Pain Mechanisms and Treatments, Ion channel regulation and function, and Neuroscience and Neuropharmacology Research.
Aβ low threshold mechanoreceptors contribute to sensory abnormalities in fibromyalgia
Passive transfer of fibromyalgia symptoms from patients to mice
Autoantibodies produce pain in complex regional pain syndrome by sensitizing nociceptors
Promiscuous G-Protein-Coupled Receptor Inhibition of Transient Receptor Potential Melastatin 3 Ion Channels by Gβγ Subunits
G protein βγ subunits inhibit TRPM3 ion channels in sensory neurons
TRPA1 activation leads to neurogenic vasodilatation: involvement of reactive oxygen nitrogen species in addition to CGRP and NO
Environmental cold exposure increases blood flow and affects pain sensitivity in the knee joints of CFA-induced arthritic mice in a TRPA1-dependent manner
TRPM8 is a neuronal osmosensor that regulates eye blinking in mice
Nociceptive Sensitizers Are Regulated in Damaged Joint Tissues, Including Articular Cartilage, When Osteoarthritic Mice Display Pain Behavior
Streptozotocin Stimulates the Ion Channel TRPA1 Directly
TRPA1 mediates the hypothermic action of acetaminophen
Monocytes expressing CX3CR1 orchestrate the development of vincristine-induced pain
TRPA1 is essential for the vascular response to environmental cold exposure
Stimulation of GLP-1 Secretion Downstream of the Ligand-Gated Ion Channel TRPA1
Modifications of Gait as Predictors of Natural Osteoarthritis Progression in STR/Ort Mice
Monoacylglycerols Activate TRPV1 – A Link between Phospholipase C and TRPV1
Methylglyoxal Evokes Pain by Stimulating TRPA1
Superoxide generation and leukocyte accumulation: key elements in the mediation of leukotriene B <sub>4</sub> ‐induced itch by transient receptor potential ankyrin 1 and transient receptor potential vanilloid 1
TRPA1 Has a Key Role in the Somatic Pro-Nociceptive Actions of Hydrogen Sulfide