Area of research
Surgery · Molecular Biology
Research interest
Research interests include Cancer research, Pancreatic cancer, Epithelial–mesenchymal transition, Downregulation and upregulation, Gene knockdown, and Medicine.
Interaction between Nitrogen, Phosphorus, and Invasive Alien Plants
The KRAS/Lin28B axis maintains stemness of pancreatic cancer cells via the let‐7i/TET3 pathway
PRMT5 promotes epithelial‐mesenchymal transition via EGFR‐β‐catenin axis in pancreatic cancer cells
Linc‐RoR promotes proliferation, migration, and invasion via the Hippo/YAP pathway in pancreatic cancer cells
The UCA1/KRAS axis promotes human pancreatic ductal adenocarcinoma stem cell properties and tumor growth.
PubMed 2019cited by 35position: middle
Analgesic effect of resveratrol on colitis-induced visceral pain via inhibition of TRAF6/NF-κB signaling pathway in the spinal cord
LncRNA UCA1 promotes migration and invasion in pancreatic cancer cells via the Hippo pathway
Long non‑coding RNA PVT1 promotes epithelial‑mesenchymal transition via the TGF‑β/Smad pathway in pancreatic cancer cells
MicroRNA-146a-5p attenuates visceral hypersensitivity through targeting chemokine CCL8 in the spinal cord in a mouse model of colitis
The mir-675-5p regulates the progression and development of pancreatic cancer via the UBQLN1-ZEB1-mir200 axis
Furin promotes epithelial-mesenchymal transition in pancreatic cancer cells via Hippo-YAP pathway
TGF-β1-miR-200a-PTEN induces epithelial–mesenchymal transition and fibrosis of pancreatic stellate cells
Chemokine CCL8 and its receptor CCR5 in the spinal cord are involved in visceral pain induced by experimental colitis in mice
Scoparone Protects Against Pancreatic Fibrosis via TGF-β/Smad Signaling in Rats
Sonic hedgehog-glioma associated oncogene homolog 1 signaling enhances drug resistance in CD44+/Musashi-1+ gastric cancer stem cells
ABCB2 (TAP1) as the downstream target of SHH signaling enhances pancreatic ductal adenocarcinoma drug resistance
Jatropholane-Type Diterpenes from <i>Euphorbia sikkimensis</i>