Area of research
Cellular and Molecular Neuroscience · Developmental Neuroscience
Research interest
Research interests include Nerve injury and regeneration, Neuroscience and Neuropharmacology Research, Neurogenesis and neuroplasticity mechanisms, and Stress Responses and Cortisol.
From early-life fluoxetine exposure to lifelong, sex-specific behavioral changes: decoding the dynamics of sensitive periods.
Fluoxetine and Ketamine Enhance Extinction Memory and Brain Plasticity by Triggering the p75 Neurotrophin Receptor Proteolytic Pathway.
Common lipidomic signatures across distinct acute brain injuries in patient outcome prediction.
Soluble Urokinase-Type Plasminogen Activator Receptor and Inflammatory Biomarker Response with Prognostic Significance after Acute Neuronal Injury - a Prospective Cohort Study.
Cleavage of the TrkB-FL receptor during epileptogenesis: insights from a kainic acid-induced model of epilepsy and human samples.
Diagnostic and prognostic performance of urine ubiquitin carboxy-terminal hydrolase L1 across multiple acute brain injury types - A longitudinal prospective cohort study.
Chronic treatment with fluoxetine regulates mitochondrial features and plasticity-associated transcriptomic pathways in parvalbumin-positive interneurons of prefrontal cortex.
The role of BDNF/TrkB Signalling in Somatostatin Neurons in Cocaine-Seeking Behaviour
Surgical Protocol for a Large, Resealable Cranial Window Enabling Longitudinal, Multi-Modal Electrophysiology Recordings of Mouse Default Mode Network
Shared metabolomic signatures for prognostic precision across brain injuries.
Shared metabolomic signatures for prognostic precision across brain injuries
TrkB transmembrane domain: bridging structural understanding with therapeutic strategy.
Rethinking the role of TRKB in the action of antidepressants and psychedelics.
A small TAT-TrkB peptide prevents BDNF receptor cleavage and restores synaptic physiology in Alzheimer's disease.
The effect of antidepressant treatment on blood BDNF levels in depressed patients: A review and methodological recommendations for assessment of BDNF in blood.
Antidepressants enter cells, organelles, and membranes.
Diagnostic and prognostic performance of urine ubiquitin carboxy-terminal hydrolase L1 across multiple acute brain injury types – A longitudinal prospective cohort study
Brain plasticity and neuroinflammatory protein biomarkers with circulating MicroRNAs as predictors of acute brain injury outcome - A prospective cohort study.
Subjective experience of the environment determines serotoninergic antidepressant treatment outcome in male mice.
Knowledge gaps in psychedelic medicalisation: Preclinical and neuroimaging mechanisms.
FDA should consider drug-therapy regimens.
Chronic treatment with fluoxetine downregulates mitochondrial activity in parvalbumin interneurons of prefrontal cortex
Brain Plasticity Modulator p75 Neurotrophin Receptor in Human Urine after Different Acute Brain Injuries-A Prospective Cohort Study.
Cleavage of the TrkB-FL Receptor During Epileptogenesis: Insights from a Kainic Acid-Induced Model of Epilepsy and Human Samples
Psychedelics promote plasticity by directly binding to BDNF receptor TrkB.
Beyond the 5-HT<sub>2A</sub> Receptor: Classic and Nonclassic Targets in Psychedelic Drug Action.
Ketamine and its metabolite 2R,6R-hydroxynorketamine promote ocular dominance plasticity and release tropomyosin-related kinase B from inhibitory control without reducing perineuronal nets enwrapping parvalbumin interneurons.
Activation of TrkB in Parvalbumin interneurons is required for the promotion of reversal learning in spatial and fear memory by antidepressants.
nNOS-induced tyrosine nitration of TRKB impairs BDNF signaling and restrains neuronal plasticity.
Antidepressants enter cells, organelles, and membranes