Area of research
Immunology · Ecology, Evolution, Behavior and Systematics
Research interest
Research interests include T-cell and Retrovirus Studies, Vector-Borne Animal Diseases, Animal Disease Management and Epidemiology, and RNA Interference and Gene Delivery.
Prognostic impact of valemetostat in relapsed/refractory adult T-cell leukaemia-lymphoma.
IL-6-driven POU2AF1 and ELL2 are key regulators of multiple myeloma-distinct transcriptional and splicing programs.
Stratagems of HTLV-1 for persistent infection and the resultant oncogenesis: Immune evasion and clonal expansion.
Evolution of primate T-cell leukemia virus type 1 accessory genes and functional divergence of its antisense proteins.
JunB-HBZ nuclear translocation by TGF-β is a key driver in HTLV-1-mediated leukemogenesis.
A Dendritic Cell-Like Transition of T Cells Is Associated with Spontaneous Remission of Adult T-Cell Leukemia-Lymphoma.
Integrative analysis of ATAC-seq and RNA-seq for cells infected by human T-cell leukemia virus type 1.
Human T-cell leukemia virus type I: modulation of viral gene expression and perturbation of host signaling pathways lead to persistent infection.
Identification of AK4 and RHOC as potential oncogenes addicted by adult T cell leukemia.
Impact of Novel Agents on Allogeneic Hematopoietic Cell Transplantation in Patients with T-Cell Lymphomas.
Impact of HTLV-1 infection on clinicopathological characteristics and tumour immune microenvironment in colorectal cancer.
Peripheral Blood CD4/CD8 Ratio Predicts Out-of-Specification Products in Lisocabtagene Maraleucel Manufacturing.
Identification of Occult Simian T-Cell Leukemia Virus Type 1 Infection in Japanese Macaques.
Validity of the criteria for clinical transformation of follicular lymphoma: analysis of a mostly biopsied cohort.
Dual resistance to cold and ethanol in <i>Penicillium</i> species persistently contaminating donated cadavers
Intra-pol proviral open region of HTLV-1 controls the transcription from both long terminal repeats
Machine learning based characterization of high risk carriers of HTLV-1-associated myelopathy (HAM).
Covalent anti-HIV compound that induces HIV-1 capsid multimerization and degradation decomposes the viral core
Identification of Natural Remission of Mother-to-Child Retroviral Transmission.
Figure S6 from HTLV-1 bZIP Factor-Induced Reprogramming of Lactate Metabolism and Epigenetic Status Promote Leukemic Cell Expansion
Figure S5 from HTLV-1 bZIP Factor-Induced Reprogramming of Lactate Metabolism and Epigenetic Status Promote Leukemic Cell Expansion
Table S4 from HTLV-1 bZIP Factor-Induced Reprogramming of Lactate Metabolism and Epigenetic Status Promote Leukemic Cell Expansion
Figure S3 from HTLV-1 bZIP Factor-Induced Reprogramming of Lactate Metabolism and Epigenetic Status Promote Leukemic Cell Expansion
Table S3 from HTLV-1 bZIP Factor-Induced Reprogramming of Lactate Metabolism and Epigenetic Status Promote Leukemic Cell Expansion
Supplementary Table S5 from A Dendritic Cell–Like Transition of T Cells Is Associated with Spontaneous Remission of Adult T-Cell Leukemia–Lymphoma
Supplementary Figure 2 from A Dendritic Cell–Like Transition of T Cells Is Associated with Spontaneous Remission of Adult T-Cell Leukemia–Lymphoma
Supplementary Figure 1 from A Dendritic Cell–Like Transition of T Cells Is Associated with Spontaneous Remission of Adult T-Cell Leukemia–Lymphoma
Data from A Dendritic Cell–Like Transition of T Cells Is Associated with Spontaneous Remission of Adult T-Cell Leukemia–Lymphoma
Supplementary Table S2 from A Dendritic Cell–Like Transition of T Cells Is Associated with Spontaneous Remission of Adult T-Cell Leukemia–Lymphoma
Table S1 from HTLV-1 bZIP Factor-Induced Reprogramming of Lactate Metabolism and Epigenetic Status Promote Leukemic Cell Expansion