Area of research
Neurology · Molecular Biology
Research interest
Research interests include Neuroblastoma Research and Treatments, Retinal Development and Disorders, Ocular Oncology and Treatments, and Cancer-related Molecular Pathways.
Protocol for retinal stress culture and single-cell analysis.
Targeting glycerophospholipid biosynthesis overcomes chemoresistance driven by SLFN11 loss in Ewing sarcoma.
Crop-OCT: a Fully Integrated Imageomics Pipeline to Identify Regional and Focal Retinopathy in Murine Models
Latent epigenetic programs in Müller glia contribute to stress and disease response in the retina.
PAX translocations remodel mitochondrial metabolism through altered leucine usage in rhabdomyosarcoma
PAX translocations remodel mitochondrial metabolism through altered leucine usage in rhabdomyosarcoma.
Bone morphogenetic protein (BMP) signaling determines neuroblastoma cell fate and sensitivity to retinoic acid
Bone morphogenetic protein (BMP) signaling determines neuroblastoma cell fate and sensitivity to retinoic acid.
A mouse model for hemoglobin SC disease recapitulates characteristic human pathologies.
<i>SLFN11</i> Loss-Induced Chemoresistance is Associated with Overexpression of Glycerophospholipid Biosynthesis in Ewing Sarcoma
Multimodal single-cell analyses reveal distinct fusion-regulated transcriptional programs in Ewing sarcoma
Multimodal single-cell analyses reveal distinct fusion-regulated transcriptional programs in Ewing sarcoma
Multimodal single-cell analyses reveal distinct fusion-regulated transcriptional programs in Ewing sarcoma.
Single cell transcriptomic profiling identifies tumor-acquired and therapy-resistant cell states in pediatric rhabdomyosarcoma.
An integrated single-cell RNA-seq map of human neuroblastoma tumors and preclinical models uncovers divergent mesenchymal-like gene expression programs
An integrated single-cell RNA-seq map of human neuroblastoma tumors and preclinical models uncovers divergent mesenchymal-like gene expression programs.
Lessons learned from 20 years of preclinical testing in pediatric cancers.
The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
The TERT Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres.
Evolutionary conservation of VSX2 super-enhancer modules in retinal development.
Preclinical Pediatric Molecular Analysis for Therapy Choice (MATCH)
Supplementary Figure S4 from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
Data from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
FIGURE 2 from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
Supplementary Figure S4 from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
Supplementary Figure S3 from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
Supplementary Figure S2 from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
Supplementary Figure S5 from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
Supplementary Figure S3 from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres
FIGURE 1 from The <i>TERT</i> Promoter is Polycomb-Repressed in Neuroblastoma Cells with Long Telomeres