Area of research
Immunology · Molecular Biology
Research interest
Research interests include Reproductive System and Pregnancy, Retinoids in leukemia and cellular processes, Endometriosis Research and Treatment, and Estrogen and related hormone effects.
TNFα-Induced Altered miRNA Expression Links to NF-κB Signaling Pathway in Endometriosis
Discovering endometriosis biomarkers with multiplex cytokine arrays
Retinoic Acid Is a Negative Regulator of sFLT1 Expression in Decidual Stromal Cells, and Its Levels Are Reduced in Preeclamptic Decidua
Curcumin attenuates proangiogenic and proinflammatory factors in human eutopic endometrial stromal cells through the NF‐κB signaling pathway
Decidual cells from women with preeclampsia exhibit inadequate decidualization and reduced sFlt1 suppression
Oral and intrauterine progestogens for atypical endometrial hyperplasia
IL-1β Inhibits Connexin 43 and Disrupts Decidualization of Human Endometrial Stromal Cells Through ERK1/2 and p38 MAP Kinase
Human endometrial stromal cell plasticity: Reversible sFlt1 expression negatively coincides with decidualization
Sustained virologic control in SIV <sup>+</sup> macaques after antiretroviral and α <sub>4</sub> β <sub>7</sub> antibody therapy
Exosomes derived from endometriotic stromal cells have enhanced angiogenic effects in vitro
Endometrial Stromal Decidualization Responds Reversibly to Hormone Stimulation and Withdrawal
Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways
A role for retinoids in human oocyte fertilization: regulation of connexin 43 by retinoic acid in cumulus granulosa cells
Estrogen increases Nrf2 activity through activation of the PI3K pathway in MCF-7 breast cancer cells
Retinoic Acid Biosynthesis Is Impaired in Human and Murine Endometriosis1
Gap junction blockade induces apoptosis in human endometrial stromal cells
Reduced connexin 43 in eutopic endometrium and cultured endometrial stromal cells from subjects with endometriosis
Analysis of Follicular Fluid Retinoids in Women Undergoing In Vitro Fertilization: Retinoic Acid Influences Embryo Quality and Is Reduced in Women With Endometriosis
Proteomic identification of neurotrophins in the eutopic endometrium of women with endometriosis
Retinoic acid suppresses growth of lesions, inhibits peritoneal cytokine secretion, and promotes macrophage differentiation in an immunocompetent mouse model of endometriosis
Retinoic acid regulates gap junction intercellular communication in human endometrial stromal cells through modulation of the phosphorylation status of connexin 43