Area of research
Molecular Biology · Cardiology and Cardiovascular Medicine
Research interest
Research interests include Biology, Cell biology, Genetics, Mesenchyme, Gene, and Medicine.
The transcriptional repressor HEY2 regulates mitochondrial oxidative respiration to maintain cardiac homeostasis
Cross-species comparison reveals that Hmga1 reduces H3K27me3 levels to promote cardiomyocyte proliferation and cardiac regeneration
TAD boundary deletion causes PITX2-related cardiac electrical and structural defects
Cardiomyocyte and endothelial cells play distinct roles in the tumour necrosis factor (TNF)-dependent atrial responses and increased atrial fibrillation vulnerability induced by endurance exercise training in mice
TBX2 specifies and maintains inner hair and supporting cell fate in the Organ of Corti
Regulation of otocyst patterning by <i>Tbx2</i> and <i>Tbx3</i> is required for inner ear morphogenesis in the mouse
Common Genetic Variants Contribute to Risk of Transposition of the Great Arteries
Lack of morphometric evidence for ventricular compaction in humans
Epigenetic and Transcriptional Networks Underlying Atrial Fibrillation
Conserved <i>NPPB</i> + Border Zone Switches From MEF2- to AP-1–Driven Gene Program
Identification of atrial fibrillation associated genes and functional non-coding variants
Loss-of-function variants in myocardin cause congenital megabladder in humans and mice
TBX2 and TBX3 act downstream of canonical WNT signaling in patterning and differentiation of the mouse ureteric mesenchyme
<i>Pitx2</i> modulates a <i>Tbx5</i> -dependent gene regulatory network to maintain atrial rhythm
Tbx2 and Tbx3 Act Downstream of Shh to Maintain Canonical Wnt Signaling during Branching Morphogenesis of the Murine Lung
A roadmap to improve the quality of atrial fibrillation management: proceedings from the fifth Atrial Fibrillation Network/European Heart Rhythm Association consensus conference
A common genetic variant within SCN10A modulates cardiac SCN5A expression
Common variants at SCN5A-SCN10A and HEY2 are associated with Brugada syndrome, a rare disease with high risk of sudden cardiac death
Tbx2 Controls Lung Growth by Direct Repression of the Cell Cycle Inhibitor Genes Cdkn1a and Cdkn1b
Inhibition of Sox2-dependent activation of <i>Shh</i> in the ventral diencephalon by Tbx3 is required for formation of the neurohypophysis
Tbx2 Terminates Shh/Fgf Signaling in the Developing Mouse Limb Bud by Direct Repression of Gremlin1
Homeobox transcription factor Pitx2: The rise of an asymmetry gene in cardiogenesis and arrhythmogenesis