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Mahshid S. Azamian

Baylor College of Medicine · US
Area of research
Genetics · Molecular Biology
Research interest
Research interests include Congenital heart defects research, Genomics and Rare Diseases, Genetics and Neurodevelopmental Disorders, and Genomic variations and chromosomal abnormalities.
h-index
32
citations
3,105
works
113
NIH funding
primary concept
email

Recent publications

Variants in the WDR44 WD40-repeat domain cause a spectrum of ciliopathy by impairing ciliogenesis initiation
Nature Communications 2024cited by 12position: middledoi
Bi-allelic variants in INTS11 are associated with a complex neurological disorder
The American Journal of Human Genetics 2023cited by 36position: middledoi
<i>SPTSSA</i> variants alter sphingolipid synthesis and cause a complex hereditary spastic paraplegia
Brain 2023cited by 34position: middledoi
TMEM161B regulates cerebral cortical gyration, Sonic Hedgehog signaling, and ciliary structure in the developing central nervous system
Proceedings of the National Academy of Sciences 2023cited by 28position: middledoi
Bi-allelic ATG4D variants are associated with a neurodevelopmental disorder characterized by speech and motor impairment
npj Genomic Medicine 2023cited by 18position: middledoi
The microRNA processor<i>DROSHA</i>is a candidate gene for a severe progressive neurological disorder
Human Molecular Genetics 2022cited by 17position: middledoi
Delineation of a novel neurodevelopmental syndrome associated with <i>PAX5</i> haploinsufficiency
Human Mutation 2022cited by 16position: middledoi
Endocannabinoid dysfunction in neurological disease: neuro-ocular DAGLA-related syndrome
Brain 2022cited by 15position: middledoi
Phenotypic expansion of CACNA1C-associated disorders to include isolated neurological manifestations
Genetics in Medicine 2021cited by 49position: middledoi
Commonalities across computational workflows for uncovering explanatory variants in undiagnosed cases
Genetics in Medicine 2021cited by 34position: middledoi
Variants in PRKAR1B cause a neurodevelopmental disorder with autism spectrum disorder, apraxia, and insensitivity to pain
Genetics in Medicine 2021cited by 27position: middledoi
Heterozygous loss-of-function variants significantly expand the phenotypes associated with loss of GDF11
Genetics in Medicine 2021cited by 26position: middledoi
De novo EIF2AK1 and EIF2AK2 Variants Are Associated with Developmental Delay, Leukoencephalopathy, and Neurologic Decompensation
The American Journal of Human Genetics 2020cited by 69position: middledoi
Biallelic MADD variants cause a phenotypic spectrum ranging from developmental delay to a multisystem disorder
Brain 2020cited by 59position: middledoi
An autosomal dominant neurological disorder caused by de novo variants in FAR1 resulting in uncontrolled synthesis of ether lipids
Genetics in Medicine 2020cited by 49position: middledoi
Clinical sites of the Undiagnosed Diseases Network: unique contributions to genomic medicine and science
Genetics in Medicine 2020cited by 42position: middledoi
Partial Loss of USP9X Function Leads to a Male Neurodevelopmental and Behavioral Disorder Converging on Transforming Growth Factor β Signaling
Biological Psychiatry 2019cited by 71position: middledoi
De Novo Variants in WDR37 Are Associated with Epilepsy, Colobomas, Dysmorphism, Developmental Delay, Intellectual Disability, and Cerebellar Hypoplasia
The American Journal of Human Genetics 2019cited by 56position: middledoi
De Novo Pathogenic Variants in N-cadherin Cause a Syndromic Neurodevelopmental Disorder with Corpus Callosum, Axon, Cardiac, Ocular, and Genital Defects
The American Journal of Human Genetics 2019cited by 51position: middledoi
Disruptive variants of <i>CSDE1</i> associate with autism and interfere with neuronal development and synaptic transmission
Science Advances 2019cited by 46position: middledoi
Bi-allelic Variants in TONSL Cause SPONASTRIME Dysplasia and a Spectrum of Skeletal Dysplasia Phenotypes
The American Journal of Human Genetics 2019cited by 37position: middledoi
IRF2BPL Is Associated with Neurological Phenotypes
The American Journal of Human Genetics 2018cited by 115position: middledoi
Biallelic Mutations in ATP5F1D, which Encodes a Subunit of ATP Synthase, Cause a Metabolic Disorder
The American Journal of Human Genetics 2018cited by 107position: middledoi
Use of Exome Sequencing for Infants in Intensive Care Units
JAMA Pediatrics 2017cited by 429position: middledoi
Recurrent Muscle Weakness with Rhabdomyolysis, Metabolic Crises, and Cardiac Arrhythmia Due to Bi-allelic TANGO2 Mutations
The American Journal of Human Genetics 2016cited by 146position: middledoi
Mutations in PURA Cause Profound Neonatal Hypotonia, Seizures, and Encephalopathy in 5q31.3 Microdeletion Syndrome
The American Journal of Human Genetics 2014cited by 111position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Jonathan Picker · Boston Children's Hospital1 papers (2022–2022)Seema R. Lalani · Baylor College of Medicine1 papers (2022–2022)Tiana M. Scott · Baylor College of Medicine1 papers (2022–2022)Stephen W. Scherer · Children's Hospital1 papers (2022–2022)Jill A. Rosenfeld · Baylor College of Medicine1 papers (2022–2022)Yoel Gofin · Texas Children's Hospital1 papers (2022–2022)W. Graf · The University of Texas Southwestern Medical Center1 papers (2022–2022)Madelyn A. Gillentine · Seattle Children's Hospital1 papers (2022–2022)Sally Ann Lynch · University College Dublin1 papers (2022–2022)Pankaj B. Agrawal · Post Graduate Institute of Medical Education and Research1 papers (2022–2022)Daryl A. Scott · Baylor College of Medicine1 papers (2022–2022) · 1 papers (2022–2022)Anne O’Donnell‐Luria · Broad Institute1 papers (2022–2022)Casie A. Genetti · Southwestern Medical Center1 papers (2022–2022)Tianyun Wang · Université Claude Bernard Lyon 11 papers (2022–2022)Rachel K. Earl · Seattle Children's Hospital1 papers (2022–2022) · 1 papers (2022–2022)Mauricio R. Delgado · Southwestern Medical Center1 papers (2022–2022)Jennifer Howe · Hospital for Sick Children1 papers (2022–2022)Stephanie DiTroia · Broad Institute1 papers (2022–2022)