Area of research
Nephrology · Clinical Biochemistry
Research interest
Research interests include Chronic Kidney Disease and Diabetes, Dialysis and Renal Disease Management, Acute Kidney Injury Research, and Advanced Glycation End Products research.
Mitochondrial oxidative damage reprograms lipid metabolism of renal tubular epithelial cells in the diabetic kidney
CD36 deletion ameliorates diabetic kidney disease by restoring fatty acid oxidation and improving mitochondrial function
Triglyceride-Glucose Index is Significantly Associated with the Risk of Hyperuricemia in Patients with Nonalcoholic Fatty Liver Disease
Berberine ameliorates renal interstitial inflammation and fibrosis in mice with unilateral ureteral obstruction
Association between kidney measurements and cognitive performance in patients with ischemic stroke
Berberine Reduces Lipid Accumulation by Promoting Fatty Acid Oxidation in Renal Tubular Epithelial Cells of the Diabetic Kidney
Effects of contralateral nephrectomy timing and ischemic conditions on kidney fibrosis after unilateral kidney ischemia-reperfusion injury
CD36 promotes NLRP3 inflammasome activation via the mtROS pathway in renal tubular epithelial cells of diabetic kidneys
Characterization of different biodegradable scaffolds in tissue engineering
The antioxidant peptide SS31 prevents oxidative stress, downregulates CD36 and improves renal function in diabetic nephropathy
Thioredoxin-interacting protein deficiency ameliorates kidney inflammation and fibrosis in mice with unilateral ureteral obstruction
<i><scp>CGB</scp>5</i> expression is independently associated with poor overall survival and recurrence‐free survival in patients with advanced gastric cancer
Mitochondria-targeted peptide SS-31 attenuates renal injury via an antioxidant effect in diabetic nephropathy
Anthocyanins inhibit high-glucose-induced cholesterol accumulation and inflammation by activating LXR&alpha; pathway in HK-2 cells
CD36 is involved in high glucose-induced epithelial to mesenchymal transition in renal tubular epithelial cells
Inhibition of c-Src/p38 MAPK pathway ameliorates renal tubular epithelial cells apoptosis in db/db mice
Knockdown of thioredoxin-interacting protein ameliorates high glucose-induced epithelial to mesenchymal transition in renal tubular epithelial cells