Area of research
Molecular Biology · Cardiology and Cardiovascular Medicine
Research interest
Research interests include Cardiovascular Function and Risk Factors, Mitochondrial Function and Pathology, Adipose Tissue and Metabolism, and Metabolism, Diabetes, and Cancer.
Intercellular Mitochondrial Transfer: Implications in Cardiovascular Health.
Introduction to the Compendium on Migration of Mitochondria Beyond Cell Boundary.
Recommendations for mitochondria transfer and transplantation nomenclature and characterization
Recommendations for mitochondria transfer and transplantation nomenclature and characterization.
Mitochondrial Targets in Ischaemic Heart Disease and Heart Failure, and their Potential for a More Efficient Clinical Translation. A Scientific Statement of the ESC Working Group on Cellular Biology of the Heart and the ESC Working Group on Myocardial Function
Expression of full-length dystrophin reverses muscular dystrophy defects in young and old mdx4cv mice
<sup>13</sup>C stable isotope tracing reveals distinct fatty acid oxidation pathways in proliferative versus oxidative cells.
Interaction of PGC-1α and GDF15 in the stressed heart.
Blunted Cardiac Mitophagy in Response to Metabolic Stress Contributes to HFpEF.
Mitochondrial dysfunction in macrophages promotes inflammation and suppresses repair after myocardial infarction
Metabolic mechanisms in physiological and pathological cardiac hypertrophy: new paradigms and challenges.
Branched-chain keto acids inhibit mitochondrial pyruvate carrier and suppress gluconeogenesis in hepatocytes
Branched-chain keto acids inhibit mitochondrial pyruvate carrier and suppress gluconeogenesis in hepatocytes
Raising NAD<sup>+</sup> Level Stimulates Short-Chain Dehydrogenase/Reductase Proteins to Alleviate Heart Failure Independent of Mitochondrial Protein Deacetylation.
Elevated MCU Expression by CaMKIIδB Limits Pathological Cardiac Remodeling.
Boosting NAD+ blunts TLR4-induced type I IFN in control and systemic lupus erythematosus monocytes
Upregulation of mitochondrial ATPase inhibitory factor 1 (ATPIF1) mediates increased glycolysis in mouse hearts
Metabolic Changes Associated With Cardiomyocyte Dedifferentiation Enable Adult Mammalian Cardiac Regeneration.
Mitochondrial interactome quantitation reveals structural changes in metabolic machinery in the failing murine heart
Diabetes Suppresses Glucose Uptake and Glycolysis in Macrophages.
Deciphering metabolic remodeling of the failing hearts.
dATP Elevation Induces Myocardial Metabolic Remodeling to Support Improved Cardiac Function
Cardiac Energy Metabolism in Heart Failure
Targeting Mitochondria-Inflammation Circuit by β-Hydroxybutyrate Mitigates HFpEF
GLUT1 overexpression enhances glucose metabolism and promotes neonatal heart regeneration
NAD<sup>+</sup> Redox Imbalance in the Heart Exacerbates Diabetic Cardiomyopathy.
Amino acid primed mTOR activity is essential for heart regeneration
Boosting mitochondrial metabolism with dietary supplements in heart failure.
Targeting Mitochondria-Inflammation Circuit by β-Hydroxybutyrate Mitigates HFpEF
A novel approach to measure mitochondrial respiration in frozen biological samples