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David T. Humphreys

Victor Chang Cardiac Research Institute ·
Area of research
Molecular Biology · Cancer Research
Research interest
Research interests include RNA Research and Splicing, RNA modifications and cancer, MicroRNA in disease regulation, and Congenital heart defects research.
h-index
33
citations
7,651
works
95
NIH funding
primary concept
email

Recent publications

Ageing limits stemness and tumorigenesis by reprogramming iron homeostasis
Nature 2024cited by 57position: middledoi
Integration mapping of cardiac fibroblast single-cell transcriptomes elucidates cellular principles of fibrosis in diverse pathologies
Science Advances 2024cited by 53position: middledoi
Standardised method for cardiomyocyte isolation and purification from individual murine neonatal, infant, and adult hearts
Journal of Molecular and Cellular Cardiology 2022cited by 23position: middledoi
Myhre syndrome is caused by dominant-negative dysregulation of SMAD4 and other co-factors
Differentiation 2022cited by 17position: middledoi
Krüppel-like factor 1 is a core cardiomyogenic trigger in zebrafish
Science 2021cited by 96position: middledoi
Sierra: discovery of differential transcript usage from polyA-captured single-cell RNA-seq data
Genome biology 2020cited by 116position: middledoi
Heterozygous loss of <i>WBP11</i> function causes multiple congenital defects in humans and mice
Human Molecular Genetics 2020cited by 26position: middledoi
A Screening Approach to Identify Clinically Actionable Variants Causing Congenital Heart Disease in Exome Data
Circulation Genomic and Precision Medicine 2018cited by 99position: middledoi
Identification of clinically actionable variants from genome sequencing of families with congenital heart disease
Genetics in Medicine 2018cited by 84position: middledoi
NAD Deficiency, Congenital Malformations, and Niacin Supplementation
New England Journal of Medicine 2017cited by 239position: middledoi
Male-lineage transmission of an acquired metabolic phenotype induced by grand-paternal obesity
Molecular Metabolism 2016cited by 166position: middledoi
Genetic variation in the two-pore domain potassium channel, TASK-1, may contribute to an atrial substrate for arrhythmogenesis
Journal of Molecular and Cellular Cardiology 2014cited by 71position: middledoi
Glioma microvesicles carry selectively packaged coding and non-coding RNAs which alter gene expression in recipient cells
RNA Biology 2013cited by 243position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Joshua W. K. Ho · Department of Health4 papers (2016–2020)Richard P. Harvey · Victor Chang Cardiac Research Institute3 papers (2018–2024)Paul Young · UNSW Sydney3 papers (2013–2022)Gavin Chapman · The University of Sydney3 papers (2018–2022)Eleni Giannoulatou · Victor Chang Cardiac Research Institute3 papers (2016–2018)Robert M. Graham · Victor Chang Cardiac Research Institute3 papers (2018–2022)Sally L. Dunwoodie · New South Wales Department of Health3 papers (2018–2022)Gillian M. Blue · The University of Sydney2 papers (2018–2018)Justin O. Szot · Prince of Wales Hospital2 papers (2018–2018)Sally A. Eaton · UNSW Sydney2 papers (2013–2016)Emma L. Duncan · King's College London2 papers (2018–2022)Eddie Ip · Victor Chang Cardiac Research Institute2 papers (2018–2018)Gary F. Sholler · The University of Sydney2 papers (2018–2018)Catherine M. Suter · The University of Sydney2 papers (2013–2016)Vaibhao Janbandhu · UNSW Sydney2 papers (2020–2024)Ralph Patrick · UNSW Sydney2 papers (2020–2024)Hartmut Cuny · New South Wales Department of Health2 papers (2018–2018)Katrina Harrison · Water Corporation of Western Australia (Australia)2 papers (2018–2018)David S. Winlaw · Northwestern University2 papers (2018–2018)Nicholas Pachter · The University of Western Australia2 papers (2018–2018)