Area of research
Immunology · Molecular Biology
Research interest
Research interests include Medicine, Endocrinology, Osteoarthritis, FOXO1, Internal medicine, and Vascular endothelial growth factor.
Nanoparticle-based inhibition of vascular endothelial growth factor receptors alleviates osteoarthritis pain and cartilage damage
Targeting Vascular Endothelial Growth Factor Receptors as a Therapeutic Strategy for Osteoarthritis and Associated Pain
Lactobacillus acidophilus Mitigates Osteoarthritis-Associated Pain, Cartilage Disintegration and Gut Microbiota Dysbiosis in an Experimental Murine OA Model
Sensory Neuron-Specific Deletion of Tropomyosin Receptor Kinase A (TrkA) in Mice Abolishes Osteoarthritis (OA) Pain via NGF/TrkA Intervention of Peripheral Sensitization
Hepatic FoxOs link insulin signaling with plasma lipoprotein metabolism through an apolipoprotein M/sphingosine-1-phosphate pathway
DOAJ (DOAJ: Directory of Open Access Journals) 2022cited by 12position: middle
Dual regulation of TxNIP by ChREBP and FoxO1 in liver
Gut-microbiota modulation: The impact of the gut-microbiota on osteoarthritis
Absence of VEGFR‐1/Flt‐1 signaling pathway in mice results in insensitivity to discogenic low back pain in an established disc injury mouse model
Pharmacological targeting of the mammalian clock reveals a novel analgesic for osteoarthritis-induced pain
Blockade of vascular endothelial growth factor receptor-1 (Flt-1), reveals a novel analgesic for osteoarthritis-induced joint pain
The Foxo1-Inducible Transcriptional Repressor Zfp125 Causes Hepatic Steatosis and Hypercholesterolemia
Development of an in vivo mouse model of discogenic low back pain
Integrated Regulation of Hepatic Lipid and Glucose Metabolism by Adipose Triacylglycerol Lipase and FoxO Proteins
FoxO1 integrates direct and indirect effects of insulin on hepatic glucose production and glucose utilization
Coupling between Nutrient Availability and Thyroid Hormone Activation
FoxO1 Inhibits Sterol Regulatory Element-binding Protein-1c (SREBP-1c) Gene Expression via Transcription Factors Sp1 and SREBP-1c