Area of research
Clinical Biochemistry · Cardiology and Cardiovascular Medicine
Research interest
Research interests include Rage (emotion), Fibrosis, Autophagy, Cardiac fibrosis, Medicine, and Heart failure.
Formononetin, a bioactive isoflavone compound in modified nuanxinkang, alleviates cardiorenal syndrome type 4-induced mitochondrial dysfunction by targeting Hippo-YAP signaling
Formononetin, a Bioactive Isoflavone Compound in Mnxk, Alleviates Crs4-Induced Mitochondrial Dysfunction by Targeting Hippo-Yap Signaling
Macrophage <scp>RAGE</scp> deficiency prevents myocardial fibrosis by repressing autophagy‐mediated macrophage alternative activation
Inhibition of AGEs-RAGE-PP2A axis alleviates cognitive impairment after chronic heart failure
AGEs–RAGE axis mediates myocardial fibrosis via activation of cardiac fibroblasts induced by autophagy in heart failure
Plantamajoside attenuates cardiac fibrosis via inhibiting <scp>AGEs activated‐RAGE</scp>/autophagy/<scp>EndMT</scp> pathway
Knockout RAGE alleviates cardiac fibrosis through repressing endothelial-to-mesenchymal transition (EndMT) mediated by autophagy
Pyroptosis and ferroptosis induced by mixed lineage kinase 3 (MLK3) signaling in cardiomyocytes are essential for myocardial fibrosis in response to pressure overload
Ablation of RAGE Attenuated Cardiac Fibrosis by Inhibition of Autophagy medicated Endothelial‐to‐mesenchymal transition
Inhibiting Receptor of Advanced Glycation End Products Attenuates Pressure Overload-Induced Cardiac Dysfunction by Preventing Excessive Autophagy