Area of research
Pulmonary and Respiratory Medicine · Physiology
Research interest
Research interests include Pulmonary hypertension, Downregulation and upregulation, Medicine, Hypoxia (environmental), Cardiology, and Internal medicine.
SARS-CoV-2 spike protein receptor-binding domain perturbates intracellular calcium homeostasis and impairs pulmonary vascular endothelial cells
Upregulation of mechanosensitive channel Piezo1 involved in high shear stress-induced pulmonary hypertension
Microbiome and metabolome dysbiosis of the gut-lung axis in pulmonary hypertension
C/EBPα promotes porcine pre-adipocyte proliferation and differentiation via mediating MSTRG.12568.2/FOXO3 trans-activation for STYX
Upregulation of Piezo1 (Piezo Type Mechanosensitive Ion Channel Component 1) Enhances the Intracellular Free Calcium in Pulmonary Arterial Smooth Muscle Cells From Idiopathic Pulmonary Arterial Hypertension Patients
NEDD9, a Hypoxia-upregulated Mediator for Pathogenic Platelet–Endothelial Cell Interaction in Pulmonary Hypertension
SESN3 Inhibited SMAD3 to Relieve Its Suppression for MiR-124, Thus Regulating Pre-Adipocyte Adipogenesis
Dysregulation of BMP9/BMPR2/SMAD signalling pathway contributes to pulmonary fibrosis and pulmonary hypertension induced by bleomycin in rats
Tetramethylpyrazine: A promising drug for the treatment of pulmonary hypertension
Mitomycin C induces pulmonary vascular endothelial‐to‐mesenchymal transition and pulmonary veno‐occlusive disease via Smad3‐dependent pathway in rats
Altered Airway Microbiota Composition in Patients With Pulmonary Hypertension
Structural and functional definition of the pulmonary vein system in a chronic hypoxia-induced pulmonary hypertension rat model
Transplantation of Mesenchymal Stem Cells Attenuates Pulmonary Hypertension by Normalizing the Endothelial-to-Mesenchymal Transition
Divergent changes of p53 in pulmonary arterial endothelial and smooth muscle cells involved in the development of pulmonary hypertension
Orai1, 2, 3 and STIM1 promote store-operated calcium entry in pulmonary arterial smooth muscle cells
Sodium tanshinone IIA sulfonate inhibits hypoxia-induced enhancement of SOCE in pulmonary arterial smooth muscle cells via the PKG-PPAR-γ signaling axis
Bortezomib alleviates experimental pulmonary hypertension by regulating intracellular calcium homeostasis in PASMCs