Cornelia Volz

ERG Alteration Due to the rd8 Mutation of the Crb1 Gene in Cln3 +/+ rd8−/rd8- Mice

Extended electrophysiological characterization of the retinal degeneration in CLN6nclf mice

Spectral Domain Optical Coherence Tomography Allows the Unification of Clinical Decision Making for the Evaluation of Choroidal Neovascularization Activity

Further Characterization of the Predominant Inner Retinal Degeneration of Aging Cln3 Δex7/8 Knock-In Mice

Suppression of SNARE‐dependent exocytosis in retinal glial cells and its effect on ischemia‐induced neurodegeneration

Mutated olfactomedin 1 in the interphotoreceptor matrix of the mouse retina causes functional deficits and vulnerability to light damage

Impact of optical coherence tomography (OCT) on decision to continue treatment for neovascuar age-related macular degeneration

Deletion of Ocular Transforming Growth Factor β Signaling Mimics Essential Characteristics of Diabetic Retinopathy

Deletion of ocular TGF-β signaling mimics essential characteristics of diabetic retinopathy

Myocilin modulates programmed cell death during retinal development

Retinal Function in Aging Homozygous Cln3 Δex7/8 Knock-In Mice

Progressive Retinal Degeneration and Glial Activation in the CLN6nclf Mouse Model of Neuronal Ceroid Lipofuscinosis: A Beneficial Effect of DHA and Curcumin Supplementation

Correction: Progressive Retinal Degeneration and Glial Activation in the CLN6^nclfMouse Model of Neuronal Ceroid Lipofuscinosis: A Beneficial Effect of DHA and Curcumin Supplementation

Curcumin, Luteolin and DHA Supplementation Abates Microglia Activation and Retinal Degeneration in the CLN6nclf Neuronal Ceroid Lipofuscinosis Mouse Model

Constitutive overexpression of Norrin activates Wnt/β-catenin and endothelin-2 signaling to protect photoreceptors from light damage

Retinal Degeneration and Microglial Activation in Mouse Models of Neuronal Ceroid Lipofuscinoses