Area of research
Epidemiology · Infectious Diseases
Research interest
Research interests include Influenza Virus Research Studies, COVID-19 Clinical Research Studies, SARS-CoV-2 and COVID-19 Research, and Respiratory viral infections research.
Glycaemic variability underlies myocyte dysfunction and myocardial injury risk in diabetes.
Inhibition of host N-myristoylation compromises the infectivity of SARS-CoV-2 due to Golgi-bypassing egress.
AVE0991, a Mas receptor agonist, increases influenza and COVID-19 severity
<i>in vivo</i>
Influenza A virus infection perturbs host cell glycosylation
Measurement of serum 1,5-AG provides insights for diabetes management and the anti-viral immune response.
Glycaemic variability underlies myocyte dysfunction and myocardial injury risk in diabetes
Increasing HbA1c is associated with reduced CD8<sup>+</sup> T cell functionality in response to influenza virus in a TCR-dependent manner in individuals with diabetes mellitus.
Whole transcriptome profiling of placental pathobiology in SARS-CoV-2 pregnancies identifies placental dysfunction signatures.
Modulation of endoplasmic reticulum stress response pathways by respiratory viruses.
Evidence for vagal sensory neural involvement in influenza pathogenesis and disease.
Host transcriptomics and machine learning for secondary bacterial infections in patients with COVID-19: a prospective, observational cohort study.
Post-acute sequelae of SARS-CoV-2 cardiovascular symptoms are associated with trace-level cytokines that affect cardiomyocyte function.
Cardiac human bitter taste receptors contain naturally occurring variants that alter function.
Nasal Delivery of Haemophilus haemolyticus Is Safe, Reduces Influenza Severity, and Prevents Development of Otitis Media in Mice.
High glycemic variability is associated with a reduced T cell cytokine response to influenza A virus
Persistent viral RNA and protein contribute to post-acute pathology.
IFI27 transcription is an early predictor for COVID-19 outcomes, a multi-cohort observational study
SARS-CoV-2 infection and viral fusogens cause neuronal and glial fusion that compromises neuronal activity.
International Pediatric COVID-19 Severity Over the Course of the Pandemic
GPR183 antagonism reduces macrophage infiltration in influenza and SARS-CoV-2 infection.
Macrophage ACE2 is necessary for SARS-CoV-2 replication and subsequent cytokine responses that restrict continued virion release.
International Pediatric COVID-19 Severity Over the Course of the Pandemic.
Transcriptomic profiling of cardiac tissues from SARS-CoV-2 patients identifies DNA damage.
Severe respiratory viral infections: T-cell functions diverging from immunity to inflammation.
The swan genome and transcriptome, it is not all black and white.
Elevated BMI reduces the humoral response to SARS-CoV-2 infection.
Cardiac human bitter taste receptors contain naturally occurring variants that alter function
The role of children in transmission of SARS-CoV-2 variants of concern within households: an updated systematic review and meta-analysis, as at 30 June 2022.
Assessing the potential for fomite transmission of SARS-CoV-2.
In vivo inhibition of nuclear ACE2 translocation protects against SARS-CoV-2 replication and lung damage through epigenetic imprinting.