Area of research
Infectious Diseases · Molecular Biology
Research interest
Research interests include Antifungal resistance and susceptibility, Gut microbiota and health, Fungal Infections and Studies, and Oral microbiology and periodontitis research.
Faecal microbiota transplant to ERadicate gastrointestinal carriage of Antibiotic-Resistant Organisms (FERARO): A feasibility randomised controlled trial
Microbiome signatures of virulence in the oral-gut-brain axis influence Parkinson’s disease and cognitive decline pathophysiology
Interplay of constipation, intestinal barrier dysfunction and fungal exposome in aetiopathogenesis of Parkinson’s disease: hypothesis with supportive data
Oral-gut microbiome interactions in advanced cirrhosis: characterisation of pathogenic enterotypes and salivatypes, virulence factors and antimicrobial resistance
Transient colonizing microbes promote gut dysbiosis and functional impairment
Modulation of the Gut Microbiota to Control Antimicrobial Resistance (AMR)—A Narrative Review with a Focus on Faecal Microbiota Transplantation (FMT)
Metabolomics analysis in saliva from periodontally healthy, gingivitis and periodontitis patients
Successful endodontic treatment reduces serum levels of cardiovascular disease risk biomarkers—high‐sensitivity C‐reactive protein, asymmetric dimethylarginine, and matrix metalloprotease‐2
Candida albicans Enhances the Progression of Oral Squamous Cell Carcinoma<i>In Vitro</i>and<i>In Vivo</i>
Candidalysins Are a New Family of Cytolytic Fungal Peptide Toxins
The impact of apical periodontitis, non‐surgical root canal retreatment and periapical surgery on serum inflammatory biomarkers
The <i>Candida albicans</i> toxin candidalysin mediates distinct epithelial inflammatory responses through p38 and EGFR-ERK pathways
Genome-scale metabolic modelling of the human gut microbiome reveals changes in the glyoxylate and dicarboxylate metabolism in metabolic disorders
Host-mycobiome metabolic interactions in health and disease
Role of Cellular Metabolism during Candida-Host Interactions
Receptor-kinase EGFR-MAPK adaptor proteins mediate the epithelial response to Candida albicans via the cytolytic peptide toxin, candidalysin
Candidalysin triggers epithelial cellular stresses that induce necrotic death
Albumin Neutralizes Hydrophobic Toxins and Modulates <i>Candida albicans</i> Pathogenicity
Abundance and diversity of resistomes differ between healthy human oral cavities and gut
New Insights in Candida albicans Innate Immunity at the Mucosa: Toxins, Epithelium, Metabolism, and Beyond
Probing the Mobilome: Discoveries in the Dynamic Microbiome
Candidalysin activates innate epithelial immune responses via epidermal growth factor receptor
Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection
Processing of <i>Candida albicans</i> Ece1p Is Critical for Candidalysin Maturation and Fungal Virulence
IL-36 and IL-1/IL-17 Drive Immunity to Oral Candidiasis via Parallel Mechanisms
Candida innate immunity at the mucosa
Oral epithelial cells orchestrate innate type 17 responses to <i>Candida albicans</i> through the virulence factor candidalysin
Candidalysin Drives Epithelial Signaling, Neutrophil Recruitment, and Immunopathology at the Vaginal Mucosa
The Role of ErbB Receptors in Infection
The Human Mucosal Mycobiome and Fungal Community Interactions