Area of research
Cardiology and Cardiovascular Medicine · Molecular Biology
Research interest
Research interests include Cardiomyopathy and Myosin Studies, Cardiovascular Effects of Exercise, Muscle Physiology and Disorders, and Viral Infections and Immunology Research.
Diagnostics of brain tumor in the early stage: current status and future perspectives
Potential cytotoxicity of truncated slow skeletal muscle troponin T (ssTnT) in a loss of function TNNT1 myopathy mouse model.
The highly conserved C-terminal end segment of troponin T binds tropomyosin and actin to function in modulating contractile kinetics.
Thin Filament Interaction and Ca
<sup>2+</sup>
-Desensitization Effect of the C-Terminal End Peptide of Cardiac Troponin T Where Loss of Function Mutations Cause Hypertrophic Cardiomyopathy
Calponin and Transgelin: Molecular Evolution, Function, Regulation and Medical Relevance
Mechanoregulation and function of calponin and transgelin.
Evolution and function of calponin and transgelin
N-terminal truncated cardiac troponin I enhances Frank-Starling response by increasing myofilament sensitivity to resting tension.
Targeting proteostasis of the HEV replicase to combat infection in preclinical models
Evolution of the N-Terminal Regulation of Cardiac Troponin I for Heart Function of Tetrapods: Lungfish Presents an Example of the Emergence of Novel Submolecular Structure to Lead the Capacity of Adaptation.
Deletion of Calponin 2 Reduces the Formation of Postoperative Peritoneal Adhesions.
Loss of Calponin 2 causes age-progressive proteinuria in mice.
Monoclonal Antibodies as Probes to Study Ligand-Induced Conformations of Troponin Subunits.
Truncation of the N-terminus of cardiac troponin I initiates adaptive remodeling of the myocardial proteosome via phosphorylation of mechano-sensitive signaling pathways.
Early Divergence of the C-Terminal Variable Region of Troponin T Via a Pair of Mutually Exclusive Alternatively Spliced Exons Followed by a Selective Fixation in Vertebrate Heart.
Troponin Variants as Markers of Skeletal Muscle Health and Diseases
Troponin Variants as Markers of Skeletal Muscle Health and Diseases.
Mechanisms of Frank-Starling law of the heart and stretch activation in striated muscles may have a common molecular origin.
A rapid degradation of calponin 2 is required for cytokinesis.
Intestinal Dysbiosis in Young Cystic Fibrosis Rabbits.
The muscle-relaxing C-terminal peptide from troponin I populates a nascent helix, facilitating binding to tropomyosin with a potent therapeutic effect.
NH<sub>2</sub>-Terminal Cleavage of Cardiac Troponin I Signals Adaptive Response to Cardiac Stressors.
Evolution of Flight Muscle Contractility and Energetic Efficiency.
The glutamic acid-rich-long C-terminal extension of troponin T has a critical role in insect muscle functions.
Production of CFTR-ΔF508 Rabbits.
High efficiency preparation of skinned mouse cardiac muscle strips from cryosections for contractility studies.
Rats genetically selected for low and high aerobic capacity exhibit altered soleus muscle myofilament functions.
The Absence of Calponin 2 in Rabbits Suggests Caution in Choosing Animal Models.
Invertebrate troponin: Insights into the evolution and regulation of striated muscle contraction.
Double deletion of calponin 1 and calponin 2 in mice decreases systemic blood pressure with blunted length-tension response of aortic smooth muscle.