Area of research
Health, Toxicology and Mutagenesis · Pediatrics, Perinatology and Child Health
Research interest
Research interests include Internal medicine, Endocrinology, Testosterone (patch), Inflammation, Chemistry, and Cell biology.
Fine Particulate Matter (PM2.5) and the Blood–Testis Barrier: An <i>in Vivo</i> and <i>in Vitro</i> Mechanistic Study
Adverse effects and potential mechanisms of polystyrene microplastics (PS-MPs) on the blood-testis barrier
Fine particulate matter (PM2.5) induces testosterone disruption by triggering ferroptosis through SIRT1/HIF-1α signaling pathway in male mice
METTL3-m6A-SIRT1 axis affects autophagic flux contributing to PM2.5-induced inhibition of testosterone production in Leydig cells
Impacts and potential mechanisms of fine particulate matter (PM<sub>2.5</sub>) on male testosterone biosynthesis disruption
Organic anion transporting polypeptide 3a1 is a novel influx pump for Perfluorooctane sulfonate in Sertoli cells and contributes to its reproductive toxicity
Perfluorooctane sulfonate induces suppression of testosterone biosynthesis via Sertoli cell-derived exosomal/miR-9-3p downregulating StAR expression in Leydig cells
The effects of fine particulate matter on the blood-testis barrier and its potential mechanisms
Perfluorooctane sulfonate interferes with non-genomic estrogen receptor signaling pathway, inhibits ERK1/2 activation and induces apoptosis in mouse spermatocyte-derived cells
PM2.5 Exposure of Mice during Spermatogenesis: A Role of Inhibitor κB Kinase 2 in Pro-Opiomelanocortin Neurons
Perfluorooctane sulfonate (PFOS) disrupts testosterone biosynthesis via CREB/CRTC2/StAR signaling pathway in Leydig cells
Glucose Homeostasis following Diesel Exhaust Particulate Matter Exposure in a Lung Epithelial Cell-Specific IKK2-Deficient Mouse Model
Concentrated Ambient PM2.5-Induced Inflammation and Endothelial Dysfunction in a Murine Model of Neural IKK2 Deficiency
Fenvalerate induces oxidative hepatic lesions through an overload of intracellular calcium triggered by the ERK/IKK/NF‐κB pathway
TNF signaling impacts glucagon-like peptide-1 expression and secretion
Exposure to Concentrated Ambient PM2.5 Compromises Spermatogenesis in a Mouse Model: Role of Suppression of Hypothalamus-Pituitary-Gonads Axis
Programming of mouse obesity by maternal exposure to concentrated ambient fine particles
Prenatal and postnatal mothering by diesel exhaust PM2.5-exposed dams differentially program mouse energy metabolism
Prenatal exposure to diesel exhaust PM<sub>2.5</sub> causes offspring β cell dysfunction in adulthood
Inactivation of TNF/LT locus alters mouse metabolic response to concentrated ambient PM2.5
From the Cover: Lung-Specific Overexpression of Constitutively Active IKK2 Induces Pulmonary and Systemic Inflammations but Not Hypothalamic Inflammation and Glucose Intolerance
Perfluorooctane sulfonate (PFOS) disrupts blood-testis barrier by down-regulating junction proteins via p38 MAPK/ATF2/MMP9 signaling pathway
Perfluorooctane sulfonate-induced testicular toxicity and differential testicular expression of estrogen receptor in male mice
Exposure to Concentrated Ambient PM<sub>2.5</sub>Shortens Lifespan and Induces Inflammation-Associated Signaling and Oxidative Stress in Drosophila
Sertoli Cell Is a Potential Target for Perfluorooctane Sulfonate–Induced Reproductive Dysfunction in Male Mice
Decreased androgen receptor expression may contribute to spermatogenesis failure in rats exposed to low concentration of bisphenol A