Area of research
Rheumatology · Epidemiology
Research interest
Research interests include Medicine, Immunology, Psoriatic arthritis, Internal medicine, Rheumatoid arthritis, and Inflammation.
Inflammatory biomarkers of atherosclerosis in SLE that may improve SLE-specific cardiovascular risk assessment tools
Real-world damage accrual in Sjögren’s disease: a 5-year multicentre GIRRCS cohort analysis
Specialized pro-resolving mediators and autoimmunity: Recent insights and future perspectives
The Relationship Between Metabolic Syndrome and Pain Catastrophizing in Psoriatic Arthritis
Patients with psoriatic arthritis and comorbid metabolic syndrome show a difficult-to-treat phenotype: another mosaic tile in the definition of a still undefined subset of patients
Association Between the Use of Oral Contraceptives and the Development of Rheumatoid Arthritis: A Systematic Review and Meta-Analysis
From pain catastrophising to multidimensional psychological distress: unravelling the complexity of difficult-to-treat psoriatic arthritis
Upadacitinib regulates pain-related pathways and BDNF expression in human monocyte-derived microglial-like cells
Clinical targeted treatment in Sjogren's disease: A systematic literature review for an evidence-based medicine approach
A Focus on Inflammatory and Bacterial Biomarkers in Secondary Peritonitis
Mediterranean diet and its low-antigenic and anti-inflammatory properties on fibromyalgia: a systematic review
POS0719 FROM PAIN CATASTROPHIZING TO MULTIDIMENSIONAL PSYCHOLOGICAL DISTRESS: UNRAVELING THE COMPLEXITY OF DIFFICULT-TO-TREAT PSORIATIC ARTHRITIS
ABS0550 MANAGEMENT OF SJOGREN'S DISEASE IN REAL LIFE: INSIGHTS FROM AN ITALIAN COHORT
ABS0953 THE RELATIONSHIP BETWEEN METABOLIC SYNDROME AND PAIN CATASTROPHIZING IN PATIENTS WITH PSORIATIC ARTHRITIS
Hopelessness is associated to severity of both digital vasculopathy and lung disease in systemic sclerosis patients: a prospective one-year study
Pain catastrophizing and its domains significantly impact rheumatoid arthritis disease activity
The role of interleukin-15 in the spectrum of inflammatory diseases
Pain catastrophizing negatively impacts drug retention rate in patients with Psoriatic Arthritis and axial Spondyloarthritis: results from a 2-years perspective multicenter GIRRCS (Gruppo Italiano di Ricerca in Reumatologia Clinica) study
Idiopathic and connective tissue disease-associated pulmonary arterial hypertension (PAH): Similarities, differences and the role of autoimmunity
Diminished expression of the ubiquitin-proteasome system in early treatment-naïve patients with rheumatoid arthritis and concomitant type 2 diabetes may be linked to IL-1 pathway hyper-activity; results from PEAC cohort
POS1148 MECHANISMS OF PAIN MODULATION BY JAK INHIBITION: UPADACITINIB REGULATES PAIN-RELATED PATHWAYS AND BDNF EXPRESSION IN MICROGLIAL CELLS
POS1248 DAMAGE IN SJÖGREN’S DISEASE: INSIGHTS FROM THE GIRRCS SJOGREN’S DISEASE COHORT
Increased Prevalence of Headaches and Migraine in Patients with Psoriatic Arthritis and Axial Spondyloarthritis: Insights from an Italian Cohort Study
AB0133 FIBROMIND: THE USE OF MINDFULNESS-BASED STRESS REDUCTION INTERVENTION AS A POTENTIAL “ADD-ON THERAPY” TO THE USUAL TREATMENTS FOR THE MANAGEMENT OF FIBROMYALGIA AND ITS IMPACT ON HOPE LEVELS
POS0854 HOPELESSNESS LEVELS ARE SIGNIFICANTLY INCREASED IN SYSTEMIC SCLEROSIS PATIENTS AND CORRELATE WITH THE RAYNAUD CONDITION SCORE AND DIGITAL ULCER SEVERITY
The specialized pro-resolving lipid mediator Protectin D1 affects macrophages differentiation and activity in Adult-onset Still’s disease and COVID-19, two hyperinflammatory diseases sharing similar transcriptomic profiles
Association between Patient Acceptable Symptom State and disease activity in psoriatic arthritis is disrupted by confounders, including comorbid fibromyalgia
Bosentan effect on echocardiographic systolic pulmonary arterial pressure in systemic sclerosis-related pulmonary hypertension: a systematic review and metanalysis
The similar expression of both ferritin and scavenger receptors activation genes in patients with COVID19 and AOSD support their role in the pathogenesis of these diseases and identify a common mechanism at the basis of the “hyperferritinemic syndromes”
Clinical correlates of a subset of anti-fibroblast antibodies in systemic sclerosis