Area of research
Cellular and Molecular Neuroscience · Endocrine and Autonomic Systems
Research interest
Research interests include Nerve injury and regeneration, Neuroscience and Neuropharmacology Research, Regulation of Appetite and Obesity, and Neurogenesis and neuroplasticity mechanisms.
A prelimbic molecular clock of protein synthesis for memory persistence
Loss of FMRP in microglia promotes degeneration of parvalbumin neurons and audiogenic seizures via progranulin insufficiency
Genetic Dissection of BDNF and TrkB Expression in Glial Cells.
Neurotrophin-3 from the dentate gyrus supports postsynaptic sites of mossy fiber-CA3 synapses and hippocampus-dependent cognitive functions.
Genetic Val66Met BDNF Variant Increases Hyperphagia on Fat-rich Diets in Mice.
Genetic dissection of BDNF and TrkB expression in glial cells
High throughput assay for compounds that boost BDNF expression in neurons.
Neurotrophin-3 from the dentate gyrus supports postsynaptic sites of mossy fiber-CA3 synapses and contextual memory
Rapid and Lasting Effects of Activating BDNF-Expressing PVH Neurons on Energy Balance.
Discrete TrkB-expressing neurons of the dorsomedial hypothalamus regulate feeding and thermogenesis.
Elevated protein synthesis in microglia causes autism-like synaptic and behavioral aberrations.
TrkB-expressing paraventricular hypothalamic neurons suppress appetite through multiple neurocircuits.
3'UTRs Regulate Mouse Ntrk2 mRNA Distribution in Cortical Neurons.
Discrete TrkB-expressing neurons of the dorsomedial hypothalamus regulate feeding and thermogenesis
TrkB-expressing neurons in the dorsomedial hypothalamus are necessary and sufficient to suppress homeostatic feeding.
Activation of Anxiogenic Circuits Instigates Resistance to Diet-Induced Obesity via Increased Energy Expenditure.
Caspase-2 promotes AMPA receptor internalization and cognitive flexibility via mTORC2-AKT-GSK3β signaling.
TrkB-expressing paraventricular hypothalamic neurons suppress appetite through multiple neurocircuits
Brain-derived neurotrophic factor is required for axonal growth of selective groups of neurons in the arcuate nucleus
Sustained expression of brain-derived neurotrophic factor is required for maintenance of dendritic spines and normal behavior
Dendritic BDNF Synthesis Is Required for Late-Phase Spine Maturation and Recovery of Cortical Responses Following Sensory Deprivation