Area of research
Molecular Biology · Cardiology and Cardiovascular Medicine
Research interest
Research interests include Cardiac electrophysiology and arrhythmias, Ion channel regulation and function, Neuroscience and Neural Engineering, and Tissue Engineering and Regenerative Medicine.
Cell-based therapies reverse the heart failure-altered right ventricular proteome towards a pre-disease state
Space microgravity improves proliferation of human iPSC-derived cardiomyocytes
Loss of cardiac myosin light chain kinase contributes to contractile dysfunction in right ventricular pressure overload
Functional and molecular effects of TNF-α on human iPSC-derived cardiomyocytes
Carfilzomib Treatment Causes Molecular and Functional Alterations of Human Induced Pluripotent Stem Cell–Derived Cardiomyocytes
Melphalan induces cardiotoxicity through oxidative stress in cardiomyocytes derived from human induced pluripotent stem cells
Machine learning identifies abnormal Ca2+ transients in human induced pluripotent stem cell-derived cardiomyocytes
Predicting Functional Responses of Progenitor Cell Exosome Potential with Computational Modeling
Cardiac Toxicity From Ethanol Exposure in Human-Induced Pluripotent Stem Cell-Derived Cardiomyocytes
The mitochondrial calcium uniporter underlies metabolic fuel preference in skeletal muscle
Aggregation of Child Cardiac Progenitor Cells Into Spheres Activates Notch Signaling and Improves Treatment of Right Ventricular Heart Failure
Targeted Elimination of Tumorigenic Human Pluripotent Stem Cells Using Suicide-Inducing Virus-like Particles
Experimental, Systems, and Computational Approaches to Understanding the MicroRNA-Mediated Reparative Potential of Cardiac Progenitor Cell–Derived Exosomes From Pediatric Patients
A human pluripotent stem cell model of catecholaminergic polymorphic ventricular tachycardia recapitulates patient-specific drug responses
Fibronectin and Cyclic Strain Improve Cardiac Progenitor Cell Regenerative Potential <i>In Vitro</i>
Knockdown of TNF-α by DNAzyme gold nanoparticles as an anti-inflammatory therapy for myocardial infarction
Identification of Therapeutic Covariant MicroRNA Clusters in Hypoxia-Treated Cardiac Progenitor Cell Exosomes Using Systems Biology
Inositol‐1,4,5‐trisphosphate induced Ca<sup>2+</sup> release and excitation–contraction coupling in atrial myocytes from normal and failing hearts