Area of research
Surgery · Molecular Biology
Research interest
Research interests include Pancreatic function and diabetes, Metabolism, Diabetes, and Cancer, Adipose Tissue and Metabolism, and Iron Metabolism and Disorders.
Larmor power limit for cyclotron radiation of relativistic particles in a waveguide
Consensus Statement on the definition and classification of metabolic hyperferritinaemia
Synergistic Inhibitory Effects of Hypoxia and Iron Deficiency on Hepatic Glucose Response in Mouse Liver
Mechanisms Linking Glucose Homeostasis and Iron Metabolism Toward the Onset and Progression of Type 2 Diabetes
Adipocyte iron regulates leptin and food intake
Metabolic aspects of high‐altitude adaptation in Tibetans
MyD88 regulates physical inactivity-induced skeletal muscle inflammation, ceramide biosynthesis signaling, and glucose intolerance
Intermittent cold exposure results in visceral adipose tissue “browning” in the plateau pika (Ochotona curzoniae)
Genetic polymorphism of APOB is associated with diabetes mellitus in sickle cell disease
A genetic mechanism for Tibetan high-altitude adaptation
Intramuscular fat and inflammation differ in older adults: The impact of frailty and inactivity
Downregulation of E3 Ubiquitin Ligases and Mitophagy-Related Genes in Skeletal Muscle of Physically Inactive, Frail Older Women: A Cross-Sectional Comparison
Dietary Iron Controls Circadian Hepatic Glucose Metabolism Through Heme Synthesis
Manganese Supplementation Protects Against Diet-Induced Diabetes in Wild Type Mice by Enhancing Insulin Secretion
Iron regulates glucose homeostasis in liver and muscle <i>via</i> AMP‐activated protein kinase in mice
Regulation of fatty acid metabolism by mTOR in adult murine hearts occurs independently of changes in PGC-1α
MitoNEET-driven alterations in adipocyte mitochondrial activity reveal a crucial adaptive process that preserves insulin sensitivity in obesity
Adipocyte iron regulates adiponectin and insulin sensitivity
Metabolic insight into mechanisms of high-altitude adaptation in Tibetans
Systemic PPARγ Deletion Impairs Circadian Rhythms of Behavior and Metabolism
Decreased serum glucose and glycosylated hemoglobin levels in patients with Chuvash polycythemia: a role for HIF in glucose metabolism