Area of research
Physiology · Cellular and Molecular Neuroscience
Research interest
Research interests include Neuropathic pain, Medicine, Spinal cord, Downregulation and upregulation, Pharmacology, and Microglia.
Amphiregulin contributes to neuropathic pain by enhancing glycolysis that stimulates histone lactylation in sensory neurons
An atlas of neuropathic pain-associated molecular pathological characteristics in the mouse spinal cord
Follistatin drives neuropathic pain in mice through IGF1R signaling in nociceptive neurons
Cytochrome P450 26A1 Contributes to the Maintenance of Neuropathic Pain
NFAT1 Orchestrates Spinal Microglial Transcription and Promotes Microglial Proliferation via c‐MYC Contributing to Nerve Injury‐Induced Neuropathic Pain
Involvement of Histone Lysine Crotonylation in the Regulation of Nerve-Injury-Induced Neuropathic Pain
C/EBPβ Participates in Nerve Trauma-Induced TLR7 Upregulation in Primary Sensory Neurons
Decoding gene expression signatures in mice trigeminal ganglion across trigeminal neuropathic pain stages via high-throughput sequencing
CXCR5 down-regulation alleviates cognitive dysfunction in a mouse model of sepsis-associated encephalopathy: potential role of microglial autophagy and the p38MAPK/NF-κB/STAT3 signaling pathway
ZNF382 controls mouse neuropathic pain via silencer-based epigenetic inhibition of <i>Cxcl13</i> in DRG neurons
MMP24 Contributes to Neuropathic Pain in an FTO-Dependent Manner in the Spinal Cord Neurons
Chemokines in chronic pain: cellular and molecular mechanisms and therapeutic potential
G protein–coupled receptor GPR151 is involved in trigeminal neuropathic pain through the induction of Gβγ/extracellular signal-regulated kinase-mediated neuroinflammation in the trigeminal ganglion
<p>Intravenous Administration of Triptonide Attenuates CFA-Induced Pain Hypersensitivity by Inhibiting DRG AKT Signaling Pathway in Mice</p>
Increased CXCL13 and CXCR5 in Anterior Cingulate Cortex Contributes to Neuropathic Pain-Related Conditioned Place Aversion
TLR8 and its endogenous ligand miR-21 contribute to neuropathic pain in murine DRG
Chemokine receptor CCR2 contributes to neuropathic pain and the associated depression via increasing NR2B-mediated currents in both D1 and D2 dopamine receptor-containing medium spiny neurons in the nucleus accumbens shell
Demethylation of G-Protein-Coupled Receptor 151 Promoter Facilitates the Binding of Krüppel-Like Factor 5 and Enhances Neuropathic Pain after Nerve Injury in Mice
Spinal CXCL9 and CXCL11 are not involved in neuropathic pain despite an upregulation in the spinal cord following spinal nerve injury
Chemokines in neuron–glial cell interaction and pathogenesis of neuropathic pain
Chemokine CCL8 and its receptor CCR5 in the spinal cord are involved in visceral pain induced by experimental colitis in mice
Promoted Interaction of C/EBPα with Demethylated <i>Cxcr3</i> Gene Promoter Contributes to Neuropathic Pain in Mice
CXCL13 drives spinal astrocyte activation and neuropathic pain via CXCR5
Chemokine CXCL13 mediates orofacial neuropathic pain via CXCR5/ERK pathway in the trigeminal ganglion of mice
Promoted Interaction of C/EBPα with Demethylated<i>Cxcr3</i>Gene Promoter Contributes to Neuropathic Pain in Mice
CXCL13/CXCR5 enhances sodium channel Nav1.8 current density via p38 MAP kinase in primary sensory neurons following inflammatory pain
Altered T-UCRs expression profile in the spinal cord of mice with neuropathic pain
Annexin A10 is involved in the development and maintenance of neuropathic pain in mice
Compound C induces the ramification of murine microglia in an AMPK-independent and small rhogtpase-dependent manner
MicroRNA-146a-5p attenuates neuropathic pain via suppressing TRAF6 signaling in the spinal cord