Area of research
Molecular Biology · Genetics
Research interest
Research interests include Signaling Pathways in Disease, Connective tissue disorders research, NF-κB Signaling Pathways, and Aortic aneurysm repair treatments.
Myeloperoxidase aggravates thoracic aortic aneurysm formation in Marfan disease.
Excessive glycosylation drives thoracic aortic aneurysm formation through integrated stress response.
Phosphatase-independent activity of smooth-muscle calcineurin orchestrates a gene expression program leading to hypertension.
Interferon stimulated gene 15 (ISG15) modulates phenotype of vascular smooth muscle cells and pathological vascular remodeling.
Versican accumulation drives Nos2 induction and aortic disease in Marfan syndrome via Akt activation.
The G4 resolvase Dhx36 modulates cardiomyocyte differentiation and ventricular conduction system development.
Resolvin D2 prevents vascular remodeling, hypercontractility and endothelial dysfunction in obese hypertensive mice through modulation of vascular and proinflammatory factors.
Integrated Stress Response Triggered by Excessive Glycosylation Drives Thoracic Aortic aneurysm
Interferon-stimulated gene 15 pathway is a novel mediator of endothelial dysfunction and aneurysms development in angiotensin II infused mice through increased oxidative stress.
Rewiring Vascular Metabolism Prevents Sudden Death due to Aortic Ruptures-Brief Report.
The NO signalling pathway in aortic aneurysm and dissection.
BMP7-based peptide agonists of BMPR1A protect the left ventricle against pathological remodeling induced by pressure overload.
Interplay between the Chd4/NuRD Complex and the Transcription Factor Znf219 Controls Cardiac Cell Identity.
Aberrant expression of miR-133a in endothelial cells inhibits angiogenesis by reducing pro-angiogenic but increasing anti-angiogenic gene expression.
Exploring the potential relationship between collagen cross-linking and impaired myocardial relaxation in Marfan syndrome: An observational study using serum biomarkers.
Extracellular Tuning of Mitochondrial Respiration Leads to Aortic Aneurysm.
Aortic disease in Marfan syndrome is caused by overactivation of sGC-PRKG signaling by NO.
CHD4 ensures stem cell lineage fidelity during skeletal muscle regeneration.
Letter by Campanero and Redondo Regarding Article, "Jugular Vein Injection of High-Titer Lentiviral Vectors Does Not Transduce the Aorta".
Aging-Associated miR-217 Aggravates Atherosclerosis and Promotes Cardiovascular Dysfunction.
Changes to the gut microbiota induced by losartan contributes to its antihypertensive effects.
Protective Effects of Short-Chain Fatty Acids on Endothelial Dysfunction Induced by Angiotensin II.
Attenuated Epigenetic Suppression of Muscle Stem Cell Necroptosis Is Required for Efficient Regeneration of Dystrophic Muscles.
NFATc3 controls tumour growth by regulating proliferation and migration of human astroglioma cells.
Cardiomyocyte calcineurin is required for the onset and progression of cardiac hypertrophy and fibrosis in adult mice.
General Statistical Framework for Quantitative Proteomics by Stable Isotope Labeling
Prostanoids in tumor angiogenesis: therapeutic intervention beyond COX-2