Area of research
Cancer Research · Oncology
Research interest
Research interests include Cancer research, Head and neck squamous-cell carcinoma, Cancer, Metastasis, Medicine, and Biology.
SMAC-armed oncolytic virotherapy enhances the anticancer activity of PD1 blockade by modulating PANoptosis
Modulation of Dendritic Cell Function via Nanoparticle-Induced Cytosolic Calcium Changes
Therapeutic Targeting of the GLS1–c-Myc Positive Feedback Loop Suppresses Glutaminolysis and Inhibits Progression of Head and Neck Cancer
ATAD3A mediates activation of RAS-independent mitochondrial ERK1/2 signaling, favoring head and neck cancer development
Blockade of glutamine-dependent cell survival augments antitumor efficacy of CPI-613 in head and neck cancer
FGF19/FGFR4 signaling axis confines and switches the role of melatonin in head and neck cancer metastasis
Circumventing AKT-Associated Radioresistance in Oral Cancer by Novel Nanoparticle-Encapsulated Capivasertib
PFKP Signaling at a Glance: An Emerging Mediator of Cancer Cell Metabolism
Simultaneously inactivating Src and AKT by saracatinib/capivasertib co-delivery nanoparticles to improve the efficacy of anti-Src therapy in head and neck squamous cell carcinoma
Histone deacetylase inhibitors suppress aggressiveness of head and neck squamous cell carcinoma via histone acetylation-independent blockade of the EGFR-Arf1 axis
Nck-associated protein 1 associates with HSP90 to drive metastasis in human non-small-cell lung cancer
ATAD3A on the Path to Cancer
FGF19 amplification reveals an oncogenic dependency upon autocrine FGF19/FGFR4 signaling in head and neck squamous cell carcinoma
Combating head and neck cancer metastases by targeting Src using multifunctional nanoparticle-based saracatinib
Autophagy blockade sensitizes human head and neck squamous cell carcinoma towards CYT997 through enhancing excessively high reactive oxygen species-induced apoptosis
Combined targeting of Arf1 and Ras potentiates anticancer activity for prostate cancer therapeutics