Cristina López

Subtyping Burkitt Lymphoma by <scp>DNA</scp> Methylation

The FBXO45–GEF-H1 Axis Controls Germinal Center Formation and B-cell Lymphomagenesis

Testicular large B‐cell lymphoma is genetically similar to PCNSL and distinct from nodal DLBCL

Discriminating activating, deactivating and resistance variants in protein kinases

Genomic landscape of follicular lymphoma across a wide spectrum of clinical behaviors

Focal structural variants revealed by whole genome sequencing disrupt the histone demethylase KDM4C in B-cell lymphomas

T‐cell prolymphocytic leukemia is associated with deregulation of oncogenic <scp>microRNAs</scp> on transcriptional and epigenetic level

Mutational mechanisms shaping the coding and noncoding genome of germinal center derived B-cell lymphomas

The genomic and transcriptional landscape of primary central nervous system lymphoma

A Novel FBXO45-Gef-H1 Axis Controls Oncogenic Signaling in B-Cell Lymphoma

Genomic and epigenomic insights into the origin, pathogenesis, and clinical behavior of mantle cell lymphoma subtypes

Acquired resistance to DZNep-mediated apoptosis is associated with copy number gains of AHCY in a B-cell lymphoma model

Genomic and transcriptomic changes complement each other in the pathogenesis of sporadic Burkitt lymphoma

JAK/STAT-Activating Genomic Alterations Are a Hallmark of T-PLL

Cryptic insertion of <i>MYC</i> exons 2 and 3 into the immunoglobulin heavy chain locus detected by whole genome sequencing in a case of “ <i>MYC</i> -negative” Burkitt lymphoma

Reconstruction of rearranged T‐cell receptor loci by whole genome and transcriptome sequencing gives insights into the initial steps of T‐cell prolymphocytic leukemia

IG-MYC+ neoplasms with precursor B-cell phenotype are molecularly distinct from Burkitt lymphomas

ACEseq – allele specific copy number estimation from whole genome sequencing

Differences between BCL2-break positive and negative follicular lymphoma unraveled by whole-exome sequencing

PROFILING OF DNA METHYLATION IN EPIDEMIOLOGICAL AND CLINICAL SUBGROUPS OF BURKITT LYMPHOMA IN THE FRAMEWORK OF THE MMML: ICGC AND BLUEPRINT CONSORTIA

B-cell–specific conditional expression of Myd88p.L252P leads to the development of diffuse large B-cell lymphoma in mice

Genes encoding members of the <scp>JAK</scp>‐<scp>STAT</scp> pathway or epigenetic regulators are recurrently mutated in T‐cell prolymphocytic leukaemia

Alterations of microRNA and microRNA-regulated messenger RNA expression in germinal center B-cell lymphomas determined by integrative sequencing analysis

DNA methylome analysis in Burkitt and follicular lymphomas identifies differentially methylated regions linked to somatic mutation and transcriptional control

MINCR is a MYC-induced lncRNA able to modulate MYC’s transcriptional network in Burkitt lymphoma cells

The <i>PCBP1</i> gene encoding poly(rc) binding protein i is recurrently mutated in <scp>B</scp>urkitt lymphoma