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Junling Gao

Sun Yat-sen University · CN
Area of research
Neurology · Epidemiology
Research interest
Research interests include Traumatic Brain Injury and Neurovascular Disturbances, Autophagy in Disease and Therapy, Intracerebral and Subarachnoid Hemorrhage Research, and Heme Oxygenase-1 and Carbon Monoxide.
h-index
28
citations
7,218
works
113
NIH funding
primary concept
email

Recent publications

Elevated miR-29a Contributes to Axonal Outgrowth and Neurological Recovery After Intracerebral Hemorrhage via Targeting PTEN/PI3K/Akt Pathway
Cellular and Molecular Neurobiology 2020cited by 18position: middledoi
<p>MicroRNA-623 Inhibits Epithelial–Mesenchymal Transition to Attenuate Glioma Proliferation by Targeting TRIM44</p>
OncoTargets and Therapy 2020cited by 12position: middledoi
Long-term chemogenetic activation of M1 glutamatergic neurons attenuates the behavioral and cognitive deficits caused by intracerebral hemorrhage
Biochemical and Biophysical Research Communications 2020cited by 6position: middledoi
Antcin C ameliorates neuronal inflammation due to cerebralhaemorrhage by inhibiting the TLR-4 pathway
Folia Neuropathologica 2020cited by 5position: middledoi
Lipoxin A4 Methyl Ester Reduces Early Brain Injury by Inhibition of the Nuclear Factor Kappa B (NF-κB)-Dependent Matrix Metallopeptidase 9 (MMP-9) Pathway in a Rat Model of Intracerebral Hemorrhage
Medical Science Monitor 2019cited by 39position: middledoi
Recombinant Osteopontin Improves Neurological Functional Recovery and Protects Against Apoptosis via PI3K/Akt/GSK-3β Pathway Following Intracerebral Hemorrhage
Medical Science Monitor 2018cited by 29position: middledoi
Vitamin D Receptor Activation Influences NADPH Oxidase (NOX<sub>2</sub>) Activity and Protects against Neurological Deficits and Apoptosis in a Rat Model of Traumatic Brain Injury
Oxidative Medicine and Cellular Longevity 2017cited by 51position: middledoi
Intraparenchymal treatment with bone marrow mesenchymal stem cell-conditioned medium exerts neuroprotection following intracerebral hemorrhage
Molecular Medicine Reports 2017cited by 22position: middledoi
Resveratrol attenuates neuronal autophagy and inflammatory injury by inhibiting the TLR4/NF-κB signaling pathway in experimental traumatic brain injury
International Journal of Molecular Medicine 2016cited by 91position: middledoi
Neuroprotective Effects of Resatorvid Against Traumatic Brain Injury in Rat: Involvement of Neuronal Autophagy and TLR4 Signaling Pathway
Cellular and Molecular Neurobiology 2016cited by 74position: middledoi
Neuroprotective effects of resveratrol against traumatic brain injury in rats: Involvement of synaptic proteins and neuronal autophagy
Molecular Medicine Reports 2016cited by 49position: middledoi
Chloroquine exerts neuroprotection following traumatic brain injury via suppression of inflammation and neuronal autophagic death
Molecular Medicine Reports 2015cited by 54position: middledoi
Neuroprotective effects of brilliant blue G on the brain following traumatic brain injury in rats
Molecular Medicine Reports 2015cited by 34position: lastdoi
PI3K/Akt/mTOR pathway participates in neuroprotection by dexmedetomidine inhibits neuronic autophagy following traumatic brain injury in rats
International Journal of Research in Medical Sciences 2014cited by 6position: middledoi
Neuroprotective effect of ceftriaxone in a rat model of traumatic brain injury
Neurological Sciences 2013cited by 82position: middledoi
Therapeutic effect of exogenous bone marrow-derived mesenchymal stem cell transplantation on silicosis via paracrine mechanisms in rats
Molecular Medicine Reports 2013cited by 38position: lastdoi
Astrocytic p-connexin 43 regulates neuronal autophagy in the hippocampus following traumatic brain injury in rats
Molecular Medicine Reports 2013cited by 32position: middledoi
Cytochrome c release in acute myocardial infarction predicts poor prognosis and myocardial reperfusion on contrast-enhanced magnetic resonance imaging
Coronary Artery Disease 2013cited by 14position: lastdoi
Attenuation of brain edema and spatial learning deficits by the inhibition of NADPH oxidase activity using apocynin following diffuse traumatic brain injury in rats
Molecular Medicine Reports 2012cited by 61position: middledoi
Effect of c-Jun NH₂-terminal kinase-mediated p53 expression on neuron autophagy following traumatic brain injury in rats.
PubMed 2012cited by 17position: middle
[Effect of expression of c-jun N-terminal kinase on neuron autophagy following diffuse brain injury in rats].
PubMed 2012cited by 3position: last

Grants

No grants ingested yet.

Frequent collaborators

Yanxia Tian · Shihezi University15 papers (2012–2020)Richard C. Wang · Zhejiang Chinese Medical University12 papers (2012–2020)Jianzhong Cui · Hebei Medical University11 papers (2012–2020)Xiaohua Jiang · People's Liberation Army 401 Hospital6 papers (2016–2020)Ying Cui · Fudan University6 papers (2013–2018)Changmeng Cui · Affiliated Hospital of Jining Medical University5 papers (2013–2017)Ran Li · Qingdao University5 papers (2012–2020)Ran Li · Hebei Medical University5 papers (2012–2018)Yan Feng · Hebei Medical University4 papers (2016–2017)Liqian Sun · Huazhong Agricultural University4 papers (2013–2015)Jian‐Zhong Cui · Tianjin University4 papers (2013–2016)Sixin Song · Academy of Medical Sciences3 papers (2012–2017)Manman Zhao · North China University of Science and Technology3 papers (2013–2020)Ying Cui · Guangzhou Medical University3 papers (2013–2020)Ying Cui · Dartmouth College3 papers (2013–2016)Minghang Li · Northwestern Polytechnical University3 papers (2016–2016) · 2 papers (2015–2016) · 2 papers (2015–2016)Wenyuan Ling · Shanghai Jiao Tong University2 papers (2020–2020)Ran Li · Guangzhou University of Chinese Medicine2 papers (2013–2016)